a MCPHS- Provider I- Ch 27 Management of Pts w/Dysrhythmias

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Dysrhythmias are disorders of   Formation/Conduction of heart’s electrical impulses  
Diagnose dysrhythmias   ECG  
Dysrhythmia naming   Site of origin, Mechanism of formation, Conduction involved  
Sympathetic stimulation vs. Parasympathetic stimulation r/t Heart rate, AV node conduction & Force of myocardial contraction   S:all increase, P:all decrease  
Chronotropy vs. Dromotropy vs. Inotropy   C:heart rate, D:AV node conduction, I:force of myocardial contraction  
# of electrodes r/t 12-lead ECG   10 w/6 on chest and 4 on limbs  
Limb electrode placement   Non-bony areas w/out major movement  
12-lead ECG reflects electrical activity in   Left ventricle  
Horizontal axis r/t ECG strip   Time & Rate  
Vertical axis r/t ECG strip   Voltage/Amplitude  
Positive deflection vs. Negative deflection   P:ECG waveform moves to top of strip, N:ECG waveform moves to bottom of strip  
P Wave r/t Atria   Atrial depolarization  
QRS Complex r/t Ventricle   Ventricular depolarization  
T Wave r/t Ventricle   Ventricular repolarization  
Atrial Repolarization r/t ECG strip   Occurs during QRS Complex  
U Wave   Repolarization of Purkinje fibers  
PR Interval   Time from SA node stimulation to conduction through AV node, Does not include ventricular depolarization  
ST Segment r/t Isoelectric line   Analyzed to determine if below/above isoelectric line  
QT Interval   Total time for ventricular depolarization and repolarization  
Prolonged QT intervals put Pt at risk for   Torsade de pointes  
Type of dysrhythmia r/t Torsade de pointes   Ventricular  
TP Interval   No electrical activity, Isoelectric line  
PP Interval vs. RR Interval   PP:determines atrial rhythm & rate, RR:determines ventricular rate & rhythm  
Each small box on ECG strip represents   .04 seconds  
If RR & PP intervals are same or < .8 seconds   Regular rhythm  
Contributing factors r/t Bradycardia   H’s and T’s  
3 H’s r/t Bradycardia   Hypovolemia, Hydrogen ions(acidosis), Hypoglycemia  
3 T’s r/t Bradycardia   Toxins, Thrombosis, Trauma  
Sinus bradycardia vs. Sinus tachycardia vs. Normal sinus rhythm   Only difference is rate  
Sinus arrhythmia r/t Respiration   Increases w/inspiration, Decreases w/expiration  
Atrial flutter r/t AV node conduction   Not all atrial impulses are conducted into ventricle, Therapeutic block at AV node  
P-wave shape r/t Atrial flutter   Saw-tooth shape  
Occurs when AV node becomes pacemaker   Junctional/Idionodal rhythm  
When P waves cannot be identified   Supraventricular tachycardia(SVT)  
SVT indicates only that rhythm is not   Ventricular tachycardia  
Emergencies r/t Dysrhythmia   Ventricular tachycardia, Ventricular fibrillation, Idioventricular rhythm, Ventricular asystole  
Ventricular fibrillation characteristics   Absence of: heartbeat, palpable pulse & respirations  
AHA guidelines r/t Unconscious adults who experience cardiac arrest d/t Ventricular fibrillation   Induce mild hypothermia for 12-24 hours  
Idioventricular rhythm   Purkinje fibers become pacemaker  
Ventricular asystole synonym   Flatline  
AV blocks occur when   AV node conduction is decreased/stopped  


   

 
 

 
 

 

 
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