click below
click below
Normal Size Small Size show me how
Stack #37210
A&P2-ClassNotes P50 / 51 Endocrine #6Test 3
| Question | Answer |
|---|---|
| ATRIAL NATRIUETIC PEPTIDE (ANP) FKA: | atrial natriuretic factor ANF |
| ANP helps with sodium | balance; produced by cells located in the R atrium |
| Kidneys produce 3 substances: | Renal erythropoietic factor, Vit. D and Renin |
| Renal Erythropoietic factor cues the: | release of erythropoietin EPO |
| EPO is produced in the: | JUXTAGLOMERULAR cells |
| CALCITRIOL - D3 - is a variation of: | Vit. D |
| Renin is produced by the: | juxtaglomerular cells for the RENIN-ANGIOTENSIN s;ystem |
| The RENIN-ANGIOTENSIN SYSTEM helps: | regulate water level of blood proteins |
| Skin produces: | CHOLECALCIFEROL,an inactive form of Vit D |
| Acromegaly is excessive: | GH production after epiphyseal growth plates close off |
| Hypo thyrodism (LO): | mental sluggishness & low metabolic rate. |
| MYXEDEMA: | hypo thyroidism in ADULTS |
| CRETINISM: | hypo thyroidism in CHILDREN |
| COLLOIDAL GOITER: | hypothyroidism caused by lack of iodine |
| HYPER THYROIDISM: (HI): | GRAVE'S Disease; Autoimmune antibodies mimic TSH & continuously stimulate release of thyroid hormone (HUGE EYE) |
| HYPER PARATHYROIDISM: | Rate, usually caused by tumors. Bones deform due to lack of calcium & their mineral salts are replaced by fibrous CT. |
| HYPO PARATHYROIDISM: | Overexcites neurons causing tetany; If untreated will lead to death |
| Addison's Disease: | Hypo secretion of mineralocorticoids & glucocortacoids. Resulting typically from adrenal neoplasms |
| Cushings Disease:i | Tumor of pituitary (usually) causes excess of cortisone production. Causes persistant hi blood sugar. |
| Hyperglycemia: AKA | DIABETES |
| Two forms of diabetes predominate: | INSIPIDUS and MILLITUS |
| INSIPIDUS IS: | deficiency of ADH from posterior pituitary |
| MILLITUS - insulin: | related problems cause high glucose levels in blood. Two types. |
| Two types of MILLITUS: | Type I and Type II |
| Type I millitus is: | juvenile onset-genetic |
| Type II millitus is: | adult onset |
| HYPOGLYCEMIA: | EXCESSIVE INSULIN SECRETION |