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Neuro5
Stack #33803
| Question | Answer |
|---|---|
| Herniation of intracrainal contects | Encephalocele |
| Only herination of meniges | meningocele |
| Herination of Meninges and brain | memingoencephalocele |
| Meninges,brain,and venticular | meningohydroencephalocele |
| Defect in posterior neuropore | myeloschisis |
| Myeloschisis always results in | Spina Bifida |
| Spina Bifida | failure of vertebral arches to form and cover spinal cord |
| Spina pinda bifida occulta | skin closed over it--unseen |
| Spinda Bifida aperta | skin not closed over it |
| As with occipilta encephoceles, | a cystic mass may accompany spina bifida |
| Caudal eminence | a cell mass cadual to neural tube, and enlarfes and cavites |
| Cudal eminence corresponds to | Secondary neurulation |
| What Secondary Neurlation process joins neural tube | caudal eminence |
| Defect of Secondary Neurulation | Myelodysplasia |
| Myelodysplasia chacteristics | usually covered with skin, and marked with unusal pigmentation, hair, telangiectases or dimple |
| Tethered cord syndrome | where the conus medullaris and filum terminale are fixed to vertebral column |
| Tethered cord syndrome causes | loss of sensation from the legs adn feets and problems with bladder control |
| 3 Primary Brain Vesicles | 1 Prosencephalon 2 Mescencephalon 3. Rhombencephalon |
| Where cervial flexure is | Rhombencephalon-Spinal cord region |
| Cephalic flexure | 2nd bend in neural tube @ mesencephalon |
| Secondary Brain vesicles | 5th---3 primarys dived into 5, and 2 more flexures |
| Secondary Brain vesicles | 1. Pontine Fleuxure, and 2. Telencephalic flexure |
| Main structure of forebrain develop | 2nd month of gestation |
| Why abnormalitis of forebrain associate with facial defects | mesoderm during 2nd month forming facial features |
| Central induction | process of froebrain development |
| End of 5th week Telencephalon gives rise to | 2 lateral expansion called Telencephalic vesicles |
| Diencephalon develops into | Thalamic nuclei AND optic cup |
| Prosencephalization | where prosencephalon develops into diencephalic and telencephalic vesicles |
| Holoprosencaphly | failure of prosencephalon to undergo cleavage |
| Alobar holoprosencephaly results in | no lobes develop, most severe large single forebrain ventricles, thalamus poorly developed, and many structures are missing |
| Hyprotelorism | close set eyes |
| Cyclops | only sinlge midline eye |
| End of 1st trimester spinal cord is | fully formed |
| Ancencephaly results in | the brain not forming, skull may be absent, and facial abnormalities |
| Types of Defects of Primary Neulation | encephalocele, myleoschisis |
| Telangiectases | large superficial cappillaries |
| Anterior neuropores closes | 4th week |
| Pontine flexure divides | divides MESO-into Myelencephalon, and metencephalon (rostral) |
| After pontine flexure what does not divide further | Mescencephalon |
| Telencephalic flexure divides the | Proencep--into diencephalon, and telencephalon (rostal) |
| Telencephalon expands into | complex lobes, and b/c largest part of brain |
| The Telencephalic VESICLE are adult derivatives of | cerbral cortex, internal capsule, ofactory bulb and tract, postions of balsal ganglia, and the amygdala, and hippocampus |
| Optic cup forms | optic nerve and retina |
| Week 10-- | major structures of CNS are recognizable |
| Immature versions of all strcutres are present by | end of 1st trimester |
| Most infants born with holoprosencephaly also have | facial abnormalities |
| Semilobar holoprosencephaly | some separtion of forebrain into 2 lobes, and partial develop of falx cerebri |
| Disease that has rudimetary but enlarged lateral and 3rd ventricles | semilobar holoprosencephaly |
| Telencephalic vesicle is AKA | cerebral hemisphers |
| The diencephalon and cerebral hemispheres forms @ | end of week 5 |