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Leech CV Test 2
Vascular disease, cardiomyopathies, rhumetic fever, and tumors
| Question | Answer |
|---|---|
| Where do berry anurysms occur | cerebral vessels |
| What can vascular disease be secondary to | injury or autoimmune response |
| True aneurysms have complete or partial wall thickness | full |
| Where is the most common place for an AAA to occur | between renal arteries and bifurcation of iliacs or in illiacs |
| what age and gender has the highest risk of AAA's | men over 50 |
| Which stage of syphlysis causes aneurysms and where do they occur | tertiary, in ascending aorta |
| In what age range and gender do aortic dissections occur | men 40-60 |
| At what size do anuerysms become extremely dangerous | 5cm |
| Which aoritc dissection is more common and where oes it occur | Type A, involves ascending aorta(Type B, distal to subclavian) |
| Pain radiating to the back and downward is indicative of what | aortic dissection |
| Does giant cell arteritis occur more in women or men | women |
| What is the most common cause of giant cell arteritis | unknown |
| focal granulomatous inflammation of the small and medium vessels of the head are indicative of which pathology | giant cell arteritis |
| headache and facial pain with possible blindness | giant cell arteritis |
| Which vessel disorder responds to steroids | giant cell arteritis and takayasu |
| early non-specific mononuclear cell infiltrate, medial fibrosis, and sometimes granulomas | Takayasu Arteritis |
| Where does Takayasu Arteritis occur | medium to large arteries; aortic arch and major branches |
| Which disease can causea loss of pulse in the upper extremeites | takayasu |
| Disease of young adults showing association with Hep B antigen | Polyarteritis Nodosa |
| Which disease is treated with immune suppresants | polyarteritis nodosa |
| segmented fibrinoid necrosis of arterial wall with nuertophilic infiltrate | polyarteritis nodosa |
| Where can polyarteritis nodosa NOT occur | lung |
| Which disease remebles PAN, but occurs in the small arteries | Microscopic Polyangitis |
| Seen in infants and children; fever, rash, lymphadenopathy, oral/conjunctival erythema | Kawasaki |
| Etiology of Kawasaki | unknwon (but lots of immuno-problems) |
| What is the triad of Wegner Granulomatosis | 1. Focal necrotizing vasculitis of lung and upper airway2. Necrotizing granulomas of upper and lower respiratory tract3. Necrotizing glomerulitis |
| C-ANCA antibodies | Wegner Granulomatosis |
| Most common in smokers | Thromboangiitis Obliterans |
| Cause of Thromboangiitis Obliterans | unknown |
| segmented thrombosis, acute, chronic, inflammation of small and medium arteries AND viens in extremeties | Thromboangiitis Obliterans |
| Which diease is secondary to athlertoslerosis, SLE, scleroderma or Buerger disease | Raynuads |
| What are vericose veins secondary to | intralunminal pressure |
| who gets varicoities more, women or men | women, especially the pregnant |
| where are most thromboplebitis and phlebothrmoviosis likely to occur | deep leg viens, caused by CHF, neoplasia, prgnancy, postop, immobilization, and local infection |
| What is Trousseau Syndrome | moving thrombophlebitis - multiple venous thrombi appearing and disappearing - hypercoaguability? |
| Superior/Inferior Vena Cava Syndrome | Superior - secondary to neoplasisms compressing the vessel INferior - same cause but neoplasms are renal or hepatic |
| What are the clinical sugns of Superior/Inferior VC syndrome | cyanosis and/or edema in the areas behind the blockage |
| Which organism is responisble for lymphaginitis | B-hemolytic strep |
Created by:
lowryc