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VN 158 pit & adrenal
| Question | Answer |
|---|---|
| ACROMEGALY | increased secretion of growth hormone |
| DIABETES INSIPIDUS - CLASSIC SIGN | DEHYDRATION - POSTURAL VS: inreased dizziness, weakness, HR |
| SIADH | DIAGNOSIS - ADMIN VASOPRESIN |
| ADDISON DISEASE | cortisol & aldosterone. They are hyponatremic. Need to regulate sodium and potassium. |
| ADDISON DISEASE - classic s/s | weakness & lethargy. Skin has hyperpigmentation (zebra) |
| CUSHING'S DISEASE - classic s/s | excess adipose tissue in abdomen. Moonface |
| GIGANTISM | hyperpituitary; excess hight |
| HYPOPHYSECTOMY | PRECAUTIONS - may develop DI so need to regulate I |
| hormone replacement | take rest of life |
| ADDISON - PRIMARY | PRIMARY ADRENAL INSUFFICIENCY due to a destructive disease process affecting the adrenal glands that results in deficiencies of CORTISOL AND ALDOSTERONE |
| ADDISON - SECONDARY | result of dysfunction of the hypothalamus or pituitary (decreased corticotropin, ACTH) which leads to decreased androgen and cortisol production |
| ADRENAL CRISIS | life threatening emergency results from a sudden marked decrease in available adrenal hormones. |
| ADRENAL CRISIS - tx | SOLUCORTEF & SOLUMEDROL IV FLUIDS |
| ANTERIOR PITUITARY GLAND - ACTH | stimulates the release of hormones from the ADRENAL CORTEX |
| ANTERIOR PITUITARY GLAND - LH | in women, stimulates the production of SEX HORMONES in ovaries during ovulation. In men, stimulates the production of testosterone in testes |
| ANTERIOR PITUITARY GLAND -FSH | in women, stimulates the production of FOLLICLE DEVELOPMENT. In Men, stimulates SPERM PRODUCTION |
| ANTERIOR PITUITARY GLAND -TSH | stimulates the release of THYROID HORMONE |
| ANTERIOR PITUITARY GLAND -GH | promotes the body's growth and development |
| ANTERIOR PITUITARY GLAND -PROLACTIN | controls milk production |
| POSTERIOR PITUITARY GLAND - VASOPRESSIN | helps control the body's water and electrolyte LEVELS |
| POSTERIOR PITUITARY GLAND - OXYTOCIN | promotes uterine contraction during labor and activates the milk ejection in nursing women |
| ADRENAL CORTEX - CORTISOL | helps control carbohydrate, protein & lipid metabolism; protects against stress |
| ADRENAL CORTEX - ALDOSTERONE | helps control the body's water and electrolyte REGULATION |
| CUSHING SYNDROME | hyperexcetion of the adrenal cortex may result in production of excess amounts of corticosteroids |
| CUSHING DISEASE | EXCESS PRODUCTION OF ACTH BECAUSE OF PITUITARY TUMOR |
| PHEOCHROMOCYTOMA | tumor of the ADRENAL MEDULLA that causes section of excessive catecholamines. Pts exhibit episoes of HTN, hypermatabolism, hyperglycemia. |
| SIADH | characterized by water imblance r/t an increase in ADH synthesis or secretion or both. Kidneys retain excessive water |
| SIMMONDS CACHEXIA | pt has muscle and organ wasting and disruption of both digestion and metabolism. |
| DIABETEST INSIPIDUS | characterized by excessive output of dilute urine |
| NEPHROGENIC DI | an inherited defect in which the renal tubules of the kidney do not respond to ADH, resulting in inadequate water reabsorption by the kidneys. |
| NEUROGENIC DI | defect in either production or excretion of ADH. From hypothalamic tumors, head trauma, infection |
| DIPSOGENIC DI | disorder of thirst stimulation. When pt ingests water, serum osmolality decreases, which causes reduced vasopressin excretion. |
Created by:
jekjes
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