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EKG''s
Question | Answer |
---|---|
normal PR measurement | <0.2 |
normal QRS interval | <0.1 |
action during PR interval | atrial depolarization |
action during QRS interval | ventricular depolarization |
action during ST segment | ventricular repolarization |
intrinsic rate of SA node/atria | 60-100 bpm |
intrinsic rate of AV node | 40-60 bpm |
intrinsic rate on ventricles | 20-40 bpm |
manifestations of sinus tach | 100-180 bpm, reg. rhythm, p waves present and look alike |
etiology of sinus tach | stress, caffeine, nicotine, fever, exercise, hyperthyroid, hypovolemia, CHF |
Tx's for sins tach | treat underlying cause, give O2, CCBs, Betablockers, digitalis |
manifestations of sinus brady | <60 bpm, reg. rhythm, P waves present and look alike |
etiology of sinus brady | drugs, MI, hyperkalemia, hypothyroid, athletes |
Tx's for sinus brady | treat underlying cause, give O2, atropine, epi, dopamine, transcutaneous pacing |
manifestations of Premature Atrial Contractions (PAC) | usually 60-100 bpm, irregular and early beat, p waves presents and look same |
cause of PAC's | stress, fatigue, alcohol, caffeine, smoking, CAD, atria enlarged, valvular dz |
Atrial dysrhythmias involve which segment | P wave |
Ventricle dysrhythmias involve this segment | QRS wave |
manifestations of atrial flutter | 220-350 bpm, atrial rhythm is regular but ventricular rhythm is either regular or irregular, P wave not identifiable, SAWTOOTH appearance |
causes of atrial flutter | cardiac dz, alcoholism, PE, pericarditis, hyperthyroidism |
Tx's for atrial flutter | anticoagulation, cardioversion, CCB, Betablockers, digitalis, valsalva, carotid massage |
manifestation of atrial fibrillation | atrial 400-600 bpm, vent. rhythms very irregular, P not identifiable, pulse deficit, decreased CO, causes pooling in atria and cause clots or emboli |
Tx's for AFib | anticoagulation, amiodarone, CCB, betas, digoxin, pacing |
when to cardiovert? | is dysrhythmia is <48hrs and unresponsive to mxs, if >48hrs treat with coumadin 3 wks before CV and 4 wks after or TEE first then anticoagulation therapy for 4 wks after CV |
PAT or PVST- paroxysmal supraventricular tach. manifestations | 150-200 bpm, QRS normal, difficult to see P waves, fast but regular rhythm |
Tx's for PVT | valsalva, carotid massage, adenosine, Bblockers, cardioversion, ablation |
causes or PSVT | CAD, MI, extreme emotions, caffeine, dig. toxicity, hypokalemia |
cause of blocks | conduction disturbance, damage to AV junction (CAD, dig. toxicity, MI, rheumatic fever) |
types of AV blocks | 1st, 2nd and 3rd degree blocks |
1st degree AV block | usually normal rate & rhythm, PR >0.2! |
2nd degree AV block/Mobitz 1/Wenckebach | atrial rate faster than ventricular, PR lengthens until QRS drops, better prognosis than type 2 |
2nd degree block/Mobitz II | atrial rate faster than ventricular, PR slightly prolonged but consistent, QRS periodically abent, may be caused by hyperkalemia |
management of 2nd degree blocks | monitor pt. for progression into higher block, if s/s then atropine, isuprel and pacemaker; withhold digoxin; if due to MI it may reverse after injury heals |
3rd degree block | A rate faster than V, P wave and QRS wave are normal lengths but completely unrelated, firing at will |
Tx of 3rd degree | treat cause, O2, pacing, atropine, epi, dopamine, prepare for CPR if goes into asystole |
bundle branch block | widened or notched QRS (bunny ears), may need pacemaker depending of how pt. tolerates the dysrhythmia |
PVC's/ premature ventricular contractions | wide and bizarre QRS, no atrial activity before QRS, often electrolyte imbalance! |
Tx for PVC | if pt. is symtomatic give lidocaine, Bblockers, O2, check electrolytes! |
ventricular tach.manifestations | looks like tombstones, 101-250 bpm, QRS > 0.12, 3 or more PVC's |
etiology of Vtach | cardiac dz, MI, dig toxicity, life threatening and may progress to Vfib |
Tx of Vtach if pt. is stable | anti-dysrhythmics (amiodarone, lidocaine, Bblockers, etc), cardioversion |
Tx of Vtach is pt. is unstable | Dfib, precordial thump, magnesium or lidocaine |
ventricular fibrillation manifestations | no P wave, erractic impulses, extremely rapid, LETHAL |
Tfib etiology | severe myocardial damage, R on T phenomenon, hypothermia, contact w/ high voltage electricity, electrolyte imbalance, med toxicity |
Txs for VFib | shock, 5 cycles of CPR then check rhythm, epi, vasopressin, amiodarone, lidocaine, magnesium |
Torsades | type of Vfib; wide then narrow spikes; hypokalemia or hypomagnesia |
PEA and asystole | flatline; PEA there is still electrical activity but no pulse, start CPR! |