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EKG''s

QuestionAnswer
normal PR measurement <0.2
normal QRS interval <0.1
action during PR interval atrial depolarization
action during QRS interval ventricular depolarization
action during ST segment ventricular repolarization
intrinsic rate of SA node/atria 60-100 bpm
intrinsic rate of AV node 40-60 bpm
intrinsic rate on ventricles 20-40 bpm
manifestations of sinus tach 100-180 bpm, reg. rhythm, p waves present and look alike
etiology of sinus tach stress, caffeine, nicotine, fever, exercise, hyperthyroid, hypovolemia, CHF
Tx's for sins tach treat underlying cause, give O2, CCBs, Betablockers, digitalis
manifestations of sinus brady <60 bpm, reg. rhythm, P waves present and look alike
etiology of sinus brady drugs, MI, hyperkalemia, hypothyroid, athletes
Tx's for sinus brady treat underlying cause, give O2, atropine, epi, dopamine, transcutaneous pacing
manifestations of Premature Atrial Contractions (PAC) usually 60-100 bpm, irregular and early beat, p waves presents and look same
cause of PAC's stress, fatigue, alcohol, caffeine, smoking, CAD, atria enlarged, valvular dz
Atrial dysrhythmias involve which segment P wave
Ventricle dysrhythmias involve this segment QRS wave
manifestations of atrial flutter 220-350 bpm, atrial rhythm is regular but ventricular rhythm is either regular or irregular, P wave not identifiable, SAWTOOTH appearance
causes of atrial flutter cardiac dz, alcoholism, PE, pericarditis, hyperthyroidism
Tx's for atrial flutter anticoagulation, cardioversion, CCB, Betablockers, digitalis, valsalva, carotid massage
manifestation of atrial fibrillation atrial 400-600 bpm, vent. rhythms very irregular, P not identifiable, pulse deficit, decreased CO, causes pooling in atria and cause clots or emboli
Tx's for AFib anticoagulation, amiodarone, CCB, betas, digoxin, pacing
when to cardiovert? is dysrhythmia is <48hrs and unresponsive to mxs, if >48hrs treat with coumadin 3 wks before CV and 4 wks after or TEE first then anticoagulation therapy for 4 wks after CV
PAT or PVST- paroxysmal supraventricular tach. manifestations 150-200 bpm, QRS normal, difficult to see P waves, fast but regular rhythm
Tx's for PVT valsalva, carotid massage, adenosine, Bblockers, cardioversion, ablation
causes or PSVT CAD, MI, extreme emotions, caffeine, dig. toxicity, hypokalemia
cause of blocks conduction disturbance, damage to AV junction (CAD, dig. toxicity, MI, rheumatic fever)
types of AV blocks 1st, 2nd and 3rd degree blocks
1st degree AV block usually normal rate & rhythm, PR >0.2!
2nd degree AV block/Mobitz 1/Wenckebach atrial rate faster than ventricular, PR lengthens until QRS drops, better prognosis than type 2
2nd degree block/Mobitz II atrial rate faster than ventricular, PR slightly prolonged but consistent, QRS periodically abent, may be caused by hyperkalemia
management of 2nd degree blocks monitor pt. for progression into higher block, if s/s then atropine, isuprel and pacemaker; withhold digoxin; if due to MI it may reverse after injury heals
3rd degree block A rate faster than V, P wave and QRS wave are normal lengths but completely unrelated, firing at will
Tx of 3rd degree treat cause, O2, pacing, atropine, epi, dopamine, prepare for CPR if goes into asystole
bundle branch block widened or notched QRS (bunny ears), may need pacemaker depending of how pt. tolerates the dysrhythmia
PVC's/ premature ventricular contractions wide and bizarre QRS, no atrial activity before QRS, often electrolyte imbalance!
Tx for PVC if pt. is symtomatic give lidocaine, Bblockers, O2, check electrolytes!
ventricular tach.manifestations looks like tombstones, 101-250 bpm, QRS > 0.12, 3 or more PVC's
etiology of Vtach cardiac dz, MI, dig toxicity, life threatening and may progress to Vfib
Tx of Vtach if pt. is stable anti-dysrhythmics (amiodarone, lidocaine, Bblockers, etc), cardioversion
Tx of Vtach is pt. is unstable Dfib, precordial thump, magnesium or lidocaine
ventricular fibrillation manifestations no P wave, erractic impulses, extremely rapid, LETHAL
Tfib etiology severe myocardial damage, R on T phenomenon, hypothermia, contact w/ high voltage electricity, electrolyte imbalance, med toxicity
Txs for VFib shock, 5 cycles of CPR then check rhythm, epi, vasopressin, amiodarone, lidocaine, magnesium
Torsades type of Vfib; wide then narrow spikes; hypokalemia or hypomagnesia
PEA and asystole flatline; PEA there is still electrical activity but no pulse, start CPR!
Created by: leh072487
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