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SFCC A&R LP3

A&R LP3

QuestionAnswer
Blood pressure cuff Non-invasive hemodynamic monitoring system we use on almost all patients each day, provides us with quantitative information concerning our patients, it reflects the pressure exerted on the wall of the arteries
Systolic blood pressure pressure of the blood in the arteries when the ventricles are contracted
Diastolic blood pressure pressure of the blood in the arteries when the ventricles are relaxed
Hemodynamic the forces which circulate blood through the body - it describes the intravascular pressure and flow that occurs when the heart muscle contracts and pumps blood throughout the body
Hemodynamic monitoring measures vascular capacities, blood volume, pump effectiveness, and tissue perfusion
Invasive monitoring performed on the seriously ill patient because it is a more accurate measurement of blood pressure, heart function, and volume status. It measures the minute to minute pressure and flow variations that occur within and between compartments.
Intravascular volume the amount of fluid circulating in the vasculature (affected by dehydration, diuresis, and volume overload due to heart or kidney failure)
Inotropy strength of myocardial contractions. Myocytes are the only muscle cells which are able to vary the strength of contraction. It can be affected by exercise, stress, pharmaceutical agents which increase the strength of myocardial contractions
Vasoactivity the expanding and contracting of blood vessels to accommodate the variations in blood flow, regulate arterial pressure, and meet metabolic demands of organs and tissues.
Hormones that affect vasoactivity Angiotensin II, epinephrine, norepinephrine, and vasopression
Chronotropy involves the timing or rate of heart contraction. This component affects tissue perfusion and is not considered a hemodynamic component. However, all four need to function normally for patients system to remain stable.
Afterload describes the resistance that the heart has to overcome during every beat to send blood into the aorta. The resistive forces include vasoactivity and blood viscosity.
Cardiac Index (CI) the amount of blood pumped by the heart per minute, per meter square of body surface area
Cardiac Output (CO) the volume of blood pumped by the heart in one minute. Normal 4-8 L/min
Central venous pressure (CVP) readings are used to approximate the right ventricular end diastolic pressure. The right ventricular end diastolic pressure assesses right ventricular flow and general fluid status. Most like the monitor used to closely watch fluid volume status.
Low CVP reflects hypovolemia or decreased venous return
High CVP reflects overhydration, increased venous return or right sided cardiac failure
Mean Arterial Pressure (MAP) reflects changes in the relationship between CO and SVR, reflects the arterial pressure in the vessels perfusing the organs (specifically the heart, brain, kidneys)
MAP equation 1 systolic plus 2 diastolic divided by 3
Low MAP indicates decreased flow to organs
High MAP indicates increased cardiac workload
Preload occurs during diastole, it is the combination of pulmonary blood filling the atria and the stretching of myocardial fibers. Preload is regulated by variability in intravascular volume.
Causes of reduced Preload sepsis, hypovolemia, and vasodilator meds (nitrates)
Volume reduction decreased preload
Volume increase increased preload, mean arterial pressure, and stroke index
Pulmonary artery pressure (PAP) blood pressure in the pulmonary artery
Increased pulmonary artery pressure may indicate a left to right cardiac shunt, pulmonary artery hypertension, COPD, emphysema, pulmonary embolus (PE), pulmonary edema, left ventricular failure
Pulmonary Capillary Wedge Pressure (PCWP or PAWP) used to approximate the left ventricular end diastolic pressure (LVEDP) left ventricular end diastolic pressure
High PCWP may indicate left ventricular failure, mitral valve pathology, cardiac insufficiency, cardiac compression post hemorrhage
Pulmonary vascular resistance (PVR) the resistance or impediment of the pulmonary vascular bed to blood flow
Increased PVR caused by pulmonary vascular disease, pulmonary embolism, or pulmonary vasculitis or hypoxia
Right ventricular pressure (RVP) direct measurement that indicates right ventricular function and general fluid status
High RVP may indicate pulmonary hypertension, right ventricle failure, congestive heart failure
Stroke index or Stroke volume index (SI or SVI) the amount of blood ejected from the heart in one cardiac cycle relative to body surface area
Increased SVI may indicate septic shock, hyperthermia, hypervolemia or be caused by medications such as dopamine, dobutamine, or digitalis
Stroke volume (SV) the amount of blood pumped by the heart per cardiac cycle, it is measured in ml/beat
Decreased stroke volume may indicate impaired cardiac contractility or valve dysfunction and may result in heart failure
Increased stroke volume may be caused by an increase in circulating volume or an increase in inotropy
Systemic Vascular resistance (SVR) the measurement of resistance or impediment of the systemic vascular bed to blood flow
Increased SVR (systemic vascular resistance) can be caused by vasoconstrictors, hypovolemia, or late septic shock
Decreased SVR (systemic vascular resistance) can be caused by early septic shock, vasodilatros, morphine, nitrates, or hypercarbia. May require medications such as Dopamine or Levophed to support your patients system.
Systolic BP normal value 90-140 mmHg
Diastolic BP normal value 60-90 mmHg
Mean arterial pressure (MAP)normal value 70-100 mmHg
Cardiac index (CI)normal value 2.5 - 4 L/min/m2 very low may indicate shock
Cardiac Output (CO) normal value 4-8 L/min
Central venous pressure (CVP) also known as right atrial pressure (RA) normal value 2-6 mmHg
Pulmonary artery pressure (PA) normal value Systolic 20-30 mmHg Diastolic 8-12 mmHg Mean 25 mmHg
Pulonary Capillary Wedge Pressure (PWCP) normal value 4-12 mmHg
Pulmonary vascular resistance (PVR) 37-250 dynes/sec/cm5 Some list 10-250
Right ventricular pressure (RV) Systolic 20-30 mmHg Diastolic 0-5 mmHg
Stroke volume (SV) 50-100 mL
Systemic vascular resistance (SVR) 800-1200 dynes/sec/cm5
Intra-arterial catheters (art lines) offer a low risk and reliable method to continuously monitor systemic blood pressure. Critically ill pts with unstable CP status benefit from continuous monitoring. A second benefit is the ability to do serial blood sampling and ABG monitoring
Increased cardiac output Increased CO may indicate high circulating volume
Decreased cardiac output decreased CO may indicate decrease in circulating volume or decrease in the strength of ventricular contractions
Created by: shellic
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