Save
Busy. Please wait.
Log in with Clever
or

show password
Forgot Password?

Don't have an account?  Sign up 
Sign up using Clever
or

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
Your email address is only used to allow you to reset your password. See our Privacy Policy and Terms of Service.


Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.
focusNode
Didn't know it?
click below
 
Knew it?
click below
Don't Know
Remaining cards (0)
Know
0:00
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

Exam 2 CCAC

ibd ibs ulcer

QuestionAnswer
Pyelonephritis and Glomerularnephritis contributing factors Urinary Stasis Foreign Bodies Anatomic Factors Functional Disorders Other
UTI clinical manifestations Chills/Fever Leukocytosis Cloudy, sedimentation Bacteremia (urine dipstick with nitrates present) Back/flank pain (Costovertebral angle figure 43-6) Nausea/vomiting Painful, frequent, urgent urination
Urine C&S is used to To determine sensitivity to antibiotics
Intravenous Pylogram or Ultrasound CT is used to? to detect obstruction
Nursing management of UTI's 3-4 L/day and careful monitoring of I & O Antimicrobial therapy and monitoring temperature and administration of antipyretics Patient teaching Pharmacological therapy Hygiene and emptying bladder Nutritional Interventions Comfort
Treated on an outpatient basis if dehydration or sepsis are absent in UTI's Antimicrobial Therapy Macrodantin and Macrobid r used to treat uncomplicated cases Ciprofloxin and Levaquin are the therapy of choice with complicated cases Gentemyacin / Ampicillian are not usually the treatment of choice because of their dosing regi
These are used for relaxing, antispasmodic therapy in UTI's Pyridium or Urised (methenamine, phenyl salicylate, methalene blue, benzic acid, atropine, and hyoscyamine)
Bacterial infection in the renal pelvis, tubules, and interstitial tissue of one or both of the kidneys Pyelonephritis
Pyelonephritis Acute Acute Enlarged kidneys with abscesses present that eventually causes atrophy and destruction of the tubules and glomeruli
Pyelonephritis Chronic Chronic Scarred kidneys from continuous damage become contracted an non-functioning – renal disease
Can be caused by bacterial infection (UTI) or functional sources that cause UTI (vesicourecteral reflux – retrograde movement of urine from the lower to upper urinary tract), an obstruction from BPH or a urinary stone Acute Pylonephritis Pathophysiology
Acute Pyelonephritis clinical manifestations Same s/s as UTI however, the vague symptoms usually subside with a few days but bacteremia and pyuria remain
Nursing/Medical Management of Acute Pyelonephritis ABX Therapy Hydration Symptom Identification
A condition where the kidney has become small, atrophied, and has lost function due to scarring and fibrosis Chronic Pyelonephritis
Chronic Pyelonephritis S Fatigue Headache Poor appetite Polyuria Excessive thirst Weight loss
Assessment/Diagnostic Findings of Chronic Pyelonephritis IV urogram Creatinine clearance, BUN and Creatinine and bacterial presence Complications ESRD HTN Kidney stones
Medical Management of Chronic Pyelonephritis Monitoring of kidney function and long-term antimicrobial therapy
Nursing Management of Chronic Pyelonephritis 3-4 L/day and careful monitoring of I & O Antimicrobial therapy and monitoring temperature and administration of antipyretics
Glomerularnephritis Inflammation of the glomerular capillaries Classified by extent of the damage, cause and extent of changes
1Glomerulonephritis Pathophysiology Immune Response where antigen-antibody complexes become trapped in the capillaries (the filtering system of the kidneys) causing an inflammatory response and IgG is detected in the capillary walls
2Glomerulonephritis Pathophysiology Immune response where antibodies react with antigens and deposit them on the glomerular basement membrane
Acute Glomerularnephritis Common in children older than 2 years of age but can occur at any age Group A beta hemolytic stept infections of the throat 2-3 weeks prior Acute viral infections of the upper respiratory tract Can be medication induced or outside antigens
Clinical Manifestations of Acute Glomerularnephritis Hematuria May or may not be visible May be cola colored from RBC and protein Edema HTN Azotemia (urea and nitrogen in the blood) Anemia
Assessment/Diagnostic Findings - Acute Glomerularnephritis H&P Renal Biopsy Urine Studies Protenuria (<3g/day) ↑ BUN ↑ Creatinine ↓ Urine output
Complications of Acute Glomerularnephritis Hypertensive encephalopathy Treatment is aimed at decreasing blood pressure without impairing renal function Heart failure Pulmonary edema
Medical Management of Acute Glomerularnephritis Treat symptoms Preserve kidney function Treat complications Dietary Management ↓ Protein ↓ Sodium
Pharmacological Treatments PCN – if streptococcal infection is present Corticosteroids Immunosuppressant therapy Loop diuretics Antihypertensive
Nursing Management of Acute Glomerularnephritis Carbohydrates to reduce catabolism of protein Patient education Diet Complications (Renal failure – fatigue, nausea & vomiting diminishing urine output or infection) Follow-up (urinalysis evaluation, blood pressure and use of medications and effective
Chronic Glomerularnephritis Caused by repeated episodes of acute Glomerularnephritis, hypertensive nephrosclerosis, hyperlipidemia, glomerular sclerosis Kidney’s decreased to 1/5 their norm siz Cortex layer shrinks Rough irregular shape& texture Renal artery r thickened Result ES
Clinical Manifestations in Chronic Glomerularnephritis Asymptomatic General symptoms Fatigue, swollen feet at night, irritability and nocturia
Chronic kidney disease symptoms Poor nourishment, yellow-gray skin, periorbital and dependent edema, retinal changes, pale mucous membranes and cardiomegalgy (heart failure), peripheral neuropathy, decreased deep tendon reflexes
Assessment/Diagnostic Findings of Chronic Glomeruonephritis Fixed specific gravity 1.010 Protenuria Urinary casts ( protein) GFR < 50mL/min Hyperkalemia Acidosis Anemia Hypoalbuminemia ↑ Phosphorus ↓ Calcium Mental status changes Impaired nerve conduction
Medical Management of Glomerularnephritis Symptom management Daily weights Caloric management HTN Diuretics High protein diet Hemodyalisis
Inflammatory Bowel Disease (IBD) Regional enteritis Crohn’s Disease Ulcerative enteritis
Facts about IBD Persons between 15-30 years of age are at greatest risk followed by persons 5-70 years of age Strong family history predisposes risk
Cause is really unknown but may be associated with Pesticides Food additives Tobacco Radiation Allergies
Crohn’s Disease---Pathophysiology Occurs in the distal ileum and ascending colon Seen in smoker’s more often than non-smoker’s Characterized by edema and thickening of mucosa & formation of ulcers & fistulas, fissures and abscesses Bowel wall thicken & intestinal lumen decreases in siz
Primary Manifestations of Chron's RLQ abdominal pain that occurs after meals Diarrhea unrelieved by defecation
Secondary Manifestations of Chron's Malnutrition/Weight loss Anemia
Chronic Manifestations of Chron's Abdominal pain Diarrhea/Steatorrhea Nutritional deficiencies/weight loss/anorexia
Barium study of the Upper GI Tract is the diagnostic method of choice and most conclusive String sign
Endoscopy, colonoscopy and intestinal biopsies help to confirm the diagnosis
Complete Blood Cell (CBC) count ↑ WBC ↓ H/H ↑ ESR ↓ Albumin ↓ Protein
Complications of Crohn’s Disease Intestinal Obstruction Abscess Fistula Enterocutaneous fistula (small bowel) Fissure Arthritis Skin lesions Conjunctivitis Oral ulcerations ↑ Risk of colon CA
Ulcerative Colitis Ulcerative and inflammatory disease of the mucosal and sub mucosal layers of the colon and rectum 5% of persons with ulcerative colitis develop colon CA
Pathophysiology of Ulcerative Colitis to Begins in the rectum and causes narrowing, shortening and thickening of the colon due to muscular hypertrophy and fat deposits Ulcerations causing bleeding Shedding of the colonic epithelium Crypt abscesses formation
Ulcerative Colitis Primary Manifestations LLQ abdominal pain Diarrhea 10-20 liquid stools per day Intermittent tetemus Rectal Bleeding
Ulcerative Colitis Secondary Manifestations Anorexia Dehydration Anemia Hypocalcemia Rebound tenderness in RLQ
Diagnostic Studies for Ulcerative Colitis Colonoscopy/Sigmoidoscopy Shortening of the colon and dilatation of the bowel loops Barium enema
Occult blood in the stool Laboratory Findings for Ulcerative Colitis ↓ H/H ↓ Albumin ↑ WBC
Complications of Ulcerative Colitis Toxic Mega colon Perforation Bleeding Vascular engorgement
Medical Management of IBD---Goal is? Reduce inflammation Provide bowel rest and healing Improving quality of life Prevention and minimization of complications
Nutritional Therapy of IBD Low-residue, high-protein, high-calorie diet Vitamin supplementation Iron replacement Adequate oral intake for fluid & electrolyte balance Avoid lactose rich and cold foods Avoid smoking Parental nutrition may be necessary
Pharmacological Therapy of IBD Aminosalicylate (Long-term maintenance) Azulfidine Sulfa-free aminosalic (Exacerbations) Asacol Antibiotics Flagyl Corticosteroids Predinsone, solu-cortef Monoclonal antibodies Remicade
Surgical Management of IBD Strictureplasty Small bowel resection Total colectomy 25% of ulcerative colitis Intestinal transplant Regional enteritis
Common Surgical Procedures of IBD Total colectomy with illiostomy Continent Ileostomy Koch Pouch Restorative protcoloectomy with ileal pouch anal anastomosis
Nursing Management of IBD Education Parental nutrition IVF Emotional support Management of an ostomy
Nursing Management of a Client with an Ileostomy Pre-Op Infusion of fluid, blood and protein ABX Low residue diet/small frequent feedings Teaching regarding life with a ileostomy
Nursing Management of a Client with an Ileostomy post op Stoma assessment/care Fecal drainage in 72 hours/indwelling catheter for a Koch pouch IVF I & O Nasogastric suction Emotional support
Mechanical obstruction Occurs from intraluminal obstruction or mural obstruction from tumors, strictures, hernias etc.
Functional obstruction Occurs from the inability to propel things along the intestinal tract like Muscular Dystrophy, DM, Parkinson’s etc.
Causes of Intestinal Obstruction Most occur in the small bowel Most common cause is adhesions Other causes: Hernias Intussusception Volvulus Paralytic ileus
Clinical Manifestations of Small Bowel Obstruction A cramping pain in the abdomen Passage of liquids or blood but no feces or flatus Vomiting Dehydration Distended abdomen Shock if untreated
Assessment/Diagnostic Findings r/t Small Bowel Obstruction Symptom related X-ray CT
Laboratory values Small obstruction CBC Electrolytes Significance of findings: Dehydration, low plasma volume and potential infection
Medical Management of Small Bowel Obstruction Decompression of the bowel through nasogastric suction (insertion of an NGT
IVF to replace ??? A, K, Cl and volume/water
Surgical intervention for small bowel obstruction Treat the underlying cause Remove a portion of the bowel
Nursing Management of Small Bowel Obstruction Maintenance of the NGT tube Output Placement Electrolyte status Return of bowel sounds
Large Bowel Obstructions Most occur beyond the splenic flexure of the colon Are not as significant as small bowel obstructions unless blood flow is disrupted which may result in strangulation or necrosis
Large Bowel Obstructions causes--- Adenocarcinoid tumors (majority) Diverticulitis IBD Benign tumors
Clinical Manifestations of Large Bowel Obstruction Symptoms progress&develop slowly Constipation only symptom if obstruction isin sigmoid colon or rectum Shape of stool is altered & may be blood weak/anorexia & weightloss Distended abdomen & marked outline of intestinal walls Crampy, lower abdominal pai
Medical Management of Large Bowel Obstruction Nasogastric decompression Restoration of fluid volume and correction of electrolyte abnormalities Colonoscopy Cecostomy with a rectal tube Surgical resection with a permanent of temporary colostomy Ilioanal anastomosis
Monitor for signs of a worsening intestinal obstruction Crampy, lower abdominal pain Absence of bowel sounds Dehydration Anemia
Appendicitis??? Occurs commonly between the age of 10-30. The most common cause of the acute abdomen Ineffective in emptying and prone to infection/inflammation becomes kinked or occluded by fecal matter, pain in the right lower quadrant and fills rapidly with pus
Umbilical or vague Epigastric pain localizing to the right lower quadrant McBurney’s Point Pain with defecation or urination due to the location of the inflammation
other clinical manifestations Nausea/Vomiting Rebound tenderness Rovsing’s sign
Rovsing’s sign is? an indication of acute appendicitis in which pressure on the left lower quadrant of the abdomen causes pain in the right lower quadrant
McBurney’s Point is??? A point above the anterior superior spine of the ilium, located on a straight line joining that process and the umbilicus, where pressure of the finger elicits tenderness in acute appendicitis
When a person has fever, nausea and pain, a????? laxative or cathartic should NEVER be given.
Diagnostic Findings for appendicitis ↑ WBC ↑ Neutrophils Diagnostic Studies X-ray CT Ultrasound
Peritonitis/Perforation complications Occurs within 24 hours of onset of pain Accompanied by fever > 37.7 degrees Celsius or 100 degrees Fahrenheit Increased abdominal pain and tenderness General appearance changes
Immediate surgery IVF Antibiotics If the appendices has already ruptured??? a drain is placed and surgery is performed after the drainage is complete.
Patient Goals Relieve pain/anxiety (Morphine Sulfate) Place in high fowler's position Reduce infection (Antibiotics) Preventing fluid & electrolyte loss or imbalance (IVF) Monitor for paralytic ileus and insert an NGT if necessary (bowel sounds) Drain care
Gallbladder Disease Cholesystitis Inflammation of the gallbladder
Gallbladder Disease Cholelithiasis Gallstones 90% of people have gallstones that have gallbladder disease
Cholecystitis Clinical mmanifestations Acute inflammation causes pain, tenderness and rigidity in the RUQ and may radiate to mid-sternal and right shoulder pain Nausea/vomiting Signs of inflammation
Causes of Cholecystitis Acalculous Occurs after major surgery or trauma without the presence of gallstones caused by alterations in fluid and electrolytes Bile stasis
Causes of Cholecystitis Calculous Causes 90% of acute cases and occurs from obstruction of the bile from a gallstone Vascular supply becomes compromised and infection occurs
Cholelithiasis ages Increased incidence in women after age 40 and affecting 30-40% of the population by age 80
Cholelithiasis risk factors Obesity Women Multiple Pregnancies Frequent changes in weight Rapid Weight loss Treatment with high dose estrogen Ileal resection Cystic Fibrosis DM
Silent No symptoms at all and found during other surgery
Symptoms of disease Occur after ingestion of fried or fatty foods including fullness, abdominal distention and pain
Symptoms of obstruction Biliary Colic
Biliary colic Pain and tenderness in the RUQ radiating to the back and right shoulder Jaundice Dark colored urine Clay colored stool Fat soluble vitamin deficiency (A, E, D and K)
Diagnostic Studies for Cholelithiasis Ultrasonography ERCP
Nutritional and Supportive Therapy Low-fat diet avoiding eggs, cream, pork, fried food, cheese, rich-dressings, gas-forming vegetables and alcohol
Non-Surgical Pharmacological – Ursodeoxycholic (UDCA)
Surgical Removal of the Gallbladder Laparoscopic Cholecystectomy Figure 40-5 Mini-Cholecyctectomy Choledochostomy Surgical Cholecyctectomy
Potential complications of gallbladder Bleeding Gastrointestinal symptoms Injury to the bowel
Pre-Operative Interventions Chest X-ray ECG Liver function tests
pre-op Teaching Coughing and deep breathing Avoid smoking for optimal post-operative health and avoiding lung complications Avoid ASA
Patient teaching for post-operative interventions NGT tube with suctioning Drainage tubes
Nursing Diagnosis for Post-Operative Patients gallbladder removal Acute pain Impaired gas exchange Impaired skin integrity Imbalanced nutrition Knowledge deficit
Nursing Interventions post op gallbladder Low Fowler's position IVF NGT Oral fluids Soft diet Turn, cough and deep breathing (splinting) and use of incentive spirometry Drain care
Patient Education post op gallbladder Activity Wound Care Resuming Eating Managing Pain Follow-up Care
morbid obesity Applied term to persons who are 2 x their ideal body weight or BMI > 30kg/M2 Basically > 100 Lbs greater than their ideal body weight
65% of people in the united States are overweight! They suffer from many potentially fatal physical health problems as well as many psychological issues related to their health and appearance
medical management for the obese Weight loss plans Medications Meridia Xenical Treating underlying medical/psychological conditions Surgical intervention
bariatric surgery selection on? Body weight and complications present History of obesity Absence of endocrine disorders Psychological stability
Psychological stability A multi-disciplinary approach of surgeon, psychiatrist, social worker, dietitian and nurse for 6-12 months prior to surgery Must be able to comply with dietary and behavioral changes after surgery
Types of Bariatric Surgery Restrictive Procedure Malabsorptive Procedure
Roux-en-Y Gastric Bypass A combined restrictive and Malabsorptive procedure for long-term weight loss. A small pouch is isolated and the jejunum is divided and a piece is attached to the new pouch.
Gastric Banding A restrictive Procedure creating a small pouch at the funds of the stomach. A silicone band is applied to the upper portion of the stomach and adjusted through laparoscopic procedures. Saline is injected to change the diameter of the band
Biliopancreatic Diversion with Duodenal Switch Stomach is removed and the jejunum is restricted from the digestive tract so fewer calories are absorbed. The ileum is divided and half attached to the duodenum and the distal end to the jejunum
Bariatric Complications of surgery Bleeding Blood Clots Bowel Obstruction Incisonal or ventral hernias Infection/Peritonitis
Pulmonary Complications of surgery Atelectasis Pneumonia
Nutritional Deficiencies of surgery Malabsorption of vitamins and minerals Iron Supplements Vitamin B12 injections due to lack of intrinsic factor
Bile Reflux Burning Epigastric pain and reflux or vomiting of bilious material that occurs with removal of the pylorus
Bile reflux treated with Questran Aluminum Hydroxide (Antacid) Reglan
Dumping Syndrome Usually occurs in clients who had Vagotomy causing a rapid emptying of the stomach contents into the jejunum which inhibits carbohydrate and electrolyte dilution in the jejunum and increases osmostic transport of extracellular fluid into the GI tract
dumping syndrome signs and symptoms S/S include: feeling of fullness, dizziness, palpitations, cramping and diarrhea/steatorrhea
nursing management dumping syndrome Monitor for potential complications 6 small feeding of 600-800 calories Frequent hydration
patient teaching Overeating causes distention and vomiting Report s/s dehydration (concentrated urine or excessive thirst) Psychosocial interventions
Nursing assessment surgery bariatric Patient and Family knowledge Nutritional Status Abdominal Assessment Post-operative Complications
Dietary Management After Surgery for bariatrics Semi-Fowler's position after eating to delay stomach emptying and dumping syndrome Antispasmodics to delay stomach emptying No fluid intake during meals; 1 hours before or after is preferred
Dietary Management After Surgery for bariatrics Meals consist of dry items and not liquid items Low-carbohydrate meals Eat smaller, more frequent meals Enteral nutrition may be necessary
Hemorrhoid info Dilated portions of the anorectal canal By 50 years of age, 50% of people have Hemmariods and can occur during pregnancy due to the pressure exerted on the tissue Caused by shearing the mucosa during defecation
Hemorrhoid types Internal Hemorrhoids External Hemorrhoids
Clinical Manifestations of Hemorrhoids Itching Burning Pain (usually with external Hemmariods) Bright-red blood with defecation
Surgical Rubber-Band Ligation surgery hemorrhoids Rubber band is placed around the tissue distal to the Hemmariods and eventually become necrotic and falls off Complications - May cause severe pain, hemorrhage and infection for some people
Cryosurgical hemorrhiodectomy surgery hemorrhoids Freezing of the Hemorrhoids to cause tissue necrosis Complications are rare but hemorrhage and abscesses can occur Main pitfall is foul-smelling discharge and increased time for wound healing
Hemarrhoidectomy surgery for hemorrhoids For advanced disease with vein thrombosis The rectal sphincter is dilated digitally and Hemmariods are removed with a clamp or cautery
patient teaching hemorrhoids Avoid straining with defecation High-Fiber diet with increased fluid intake Good personal hygiene
Pharmacological Interventions for hemorrhoids Psyllium (Metamucil)
further treatment for hemorrhoids Warm sitz bath Analgesic ointments/suppositories and astringents Bed rest
indirect hernia abdominal intestine may remain within the inquinal canal or extrude past the external ring
indirect hernia is the? most common type of hernia
indirect hernia is located? located within the femoral canal
indirect hernia assessment appears as swelling. During palpation, have the patient cough. You will feel pressure against your finger tip. Palpate the soft mass
inguinal hernias??? extrusion of the abdominal intestine into the inquinal ring. Bulging occurs in the area around the pubis. Abdominal intestine may remain withing the inqunal ring or extrude past the external ring.
inquinal hernia sign and symptoms most often painless, appears as swelling, while palpating, have the patient cough, you will feel pressure on your finger tips
femoral hernia is??? A bulge that occurs over the area of the femoral artery. The right side is affected more than the left. This is the least common of the 3 hernias
femoral hernia S & S MAy not be painfu;, but if strangulation occurs, there will be a lot of pain
The colorectal area includes the rectum and the colon Andenocarcinoma 95%
Colorectal Cancer is the 3rd most common cause of cancer deaths in U.S Incidence increases with age (>85 years old) Family history of colon cancer History of irritable bowel disease/polyps A disease of Western cultures
Polyps are Mass of tissue arising from bowel wall; protrudes into lumen Most often occur in sigmoid and rectum 30% of people over 50 have polyps Most are benign but some have potential to become malignant (< 1% become malignant but all colorectal cancers arise from these polyps)
Polyps Manifestations Most asymptomatic Intermittent painless rectal bleeding is most common presenting symptom
Polyps diagnostic findings Diagnosis is based on colonoscopy Most reliable since allows inspection of entire colon with biopsy or polypectomy if indicated Repeat every 3 years since polyps recur
Symptoms are present based on: Location of the cancer Stage of the disease Function of the respective parts Change in bowel habits/patterns Blood in the stool
related polyps symptoms Anemia Anorexia Weight loss Fatigue
Lesions signs and symtoms Tenesmus (ineffective, painful straining) Rectal Pain Feeling of incomplete evacuation Alternating constipation/Diarrhea Bloody stool
right sided lesions Dull abdominal pain Melena Weakness Fatigue
Left sided lesions Bright red blood in the stool Obstruction (s/s) Abdominal pain Cramping Narrow ribbon-like stools Constipation/ Diarrhea Distention
Diagnostic findings for lesions Abdominal & rectal examination Fecal occult blood testing Barium enema Proctosigmoidoscopy Colonoscopy (Most definitive) Biopsy Carcinoembryonic antigen (CEA)
Lesion complications Partial/complete bowel obstruction Ulceration/Hemorrhage Perforation Abscess Peritonitis Sepsis Shock
Gerontological Consideration Incidence increases with age Common in women secondary to breast cancer and secondary to men only in prostrate and lung cancer
Gerontological Consideration Dietary carcinogens Lack of fiber Excess fat intake Alcohol consumption Smoking
Medical Management Dependent on the stage of the disease Dukes’ Classification
Dukes’ Classification a&B Class A – tumor of muscular mucosa and submucosa Class B1 – tumor extends into the mucosa Class B2 – tumor extends through the bowel wall into fat with no nodular involvement
Dukes’ Classification C&D Class C1 – Nodular involvement but tumor is limited to the bowel wall Class C2 – Nodular involvement and tumor extends through the bowel wall Class D – Advanced with metastasis
supportive therapy 5-Flourouacil and Pelvic radiation (Class B or C) Mitocyin
Adjunctive Therapy Radiation/Chemotherapy Inoperable tumors are given radiation Intracavitary devices
Palliative care is used in Class D and can involve surgery or radiation for alleviation of symptoms
Surgery for lesions Segmental Resection with anastomosis (tumor and surrounding tissues with blood vessels and lymph nodes)
surgery for lesions Abdominoperineal Resection with permanent colostomy (removal of the tumor and portion of the sigmoid colon and the rectal and anal sphincter
surgical management Temporary Colostomy and segmental resection and anastomosis of the colostomy Permanent colostomy or illiostomy for obstruction lesions
surgical management of J pouch J Pouch (2 step process that first involves and temporary illiostomy loop to direst stool to the J pouch and then reversal of the illiostomy
Ileostomy Ileostomy: opening from ileum through abdominal wall; for surgical treatment of ulcerative colitis and Crohn’s
Cecostomy: Cecostomy: opening between cecum & abdominal wall; uncommon; temporary; used before surgery or for palliation
Colostomy: : opening between colon & abdominal wall: Temporary: located in transverse colon Loop & double barrel temporary but can be permanent
Gerontological Considerations Surgical care of the colostomy Skin care Complications to the stoma from decreased blood flow from arteriosclerosis
Care Planning for Colorectal Cancer S S/S (Fatigue, abdominal or rectal pain, past and present elimination patterns, characteristics of stool)
Care Planning for Colorectal Cancer Health History History (Colorectal cancer, polyps, IBD) Medication review Dietary habits (including noted weight loss) Auscultation of bowel sounds Palpation Evaluation of stool specimens
Nursing Diagnosis for Colorectal Cancer Imbalanced Nutrition, less than body requirements Risk for fluid volume deficit Anxiety Risk for ineffective therapeutic regime Impaired skin integrity Disturbed body image Ineffective sexuality patterns
Colorectal Cancer complications Intraperitoneal infection Complete large bowel obstruction GI bleeding Perforation Peritonitis Abscess Sepsis
Planning and goals for Colorectal Cancer Nutrition Adequate hydration Knowledge regarding surgical procedure, self-care, maintenance of the ostomy Skin care (recognition and treatment of irritated areas
Ostomy Surgery Preparation Preop preparation Psychological preparation ET nurse consult (mark stoma site preop) United Ostomy Association
Bowel prep ostomy surgery pre-op Osmotic lavage (Go-Lytely) Nonabsorbable neomycin & erythomycin orally to decrease number of intracolonic bacteria
Nursing care ostomy Osmotic lavage (Go-Lytely) Nonabsorbable neomycin & erythomycin orally to decrease number of intracolonic bacteria
stoma assessment Stoma has a beefy red appearance If blood flow is restricted, it will appear light pink or grey Skin should be clean, dry and without irritation/ appliance should be fitted to the stoma without strangling the stoma
Nursing Management of Chronic Glomerularnephritis Patient Teaching Compliance with follow-up care and treatment HD care (procedure, how to care for the access site, dietary restrictions and lifestyle modifications) Family involvement and electrolyte imbalance
Nephrotic Syndrome Increased protein in the urine Protenuria Especially albumin Hypoalbuminemia Decreased albumin in the blood Edema Hyperlipidemia Increased serum cholesterol
Clinical Manifestations of Nephrotic Syndrome Edema - soft, pitting periorbital edema and dependent edema (sacrum, ankles, hands and abdomen), anasarca and ascites Irritability Headache Malaise
Assessment/Diagnostic Findings of Nephrotic Syndrome Protenuria >3.5 g/day – Hallmark diagnostic sign ↑ Albumin Needle biopsy
Medical Management of Nephrotic Syndrome Goal Preserve renal function Prevent complications Dietary Management ↓ Protein ↓ Cholesterol ↓ Sodium
Pharmacological Interventions Diuretics ACE inhibitors with combination loop-diuretics Antineoplastic agents and immunosuppressant Corticosteroids and NSAIDS
Nursing Management of Nephrotic Syndrome Assessment and treatment of edema Patient Teaching Dietary Management Medication compliance Infections Follow-up with medical personnel
Testicular Cancer Most common cancer in men age 15 to 40 Highly treatable and curable
Testicular Cancer risk faxtors : undescended testicles, positive family history, cancer of one testicle, Caucasian American race
Testicular Cancer manifestation painless lump or mass in the testes
Early diagnosis Testicular Cancer : monthly testicular self-exam (TSE) and annual testicular exam
Testicular Cancer Treatment: orchidectomy, retroperitoneal lymph node dissection (open or laparoscopic), radiation therapy, and chemotherapy
Testicular Cancer Nursing Management Assess physical and psychological status Support coping ability Address issues of body image and sexuality Encourage a positive attitude Provide patient teaching Provide TSE and follow-up care
Factors involved in stone formation: Factors involved in stone formation Diet, metabolism, genes, climate, lifestyle, and occupation
Incidence of kidney stones Occurs in Caucasian persons 30-50 years of age Occurs most often in men Reoccurrence happens in 50% of cases
Urolithiasis Renal calculi or “stones” in the urinary tract
Nephrolithiasis Renal calculi or “stones” in the kidney
Kidney stone Patho 1 Form in the urinary tract when levels of calcium oxalate, calcium phosphate and uric acid increase. Can be caused by obstruction Stones can vary in size
Kidney stone Patho 2 Several theories regarding formation including lack of substances to dehydration Related to increased calcium concentrates in the blood
Types of Stones Calcium Phosphate Calcium Oxalate Uric Acid Cystine Stuvite
Edications that cause kidney stones Antacids, Diamox, Vit D, Laxatives, High doses of ASA
Increased uric acid level causing kidney stones Uric acid stones occur in patients with gout and Myleoproliferative disorders
Increased stuvite level causing kidney stones Struvite stones are caused by ammonia rich urine such as with infection with ureasesplitting bacteria
Increased cystine levels causing kidney stones Cystine stones occur with patients who have inherited defects in renal absorption
Clinical Manifestations of kidney stones Dependent on the cause: Obstruction Infection Edema Classic S/S Pain Abdominal or flank pain and does not reflect the size of the stone. Radiates to the genitals, thigh and groin Hematuria
diagnostic findings of kidney stones Confirmed on X-ray Ultrasonography Blood work 24 hour urine Calcium Uric acid Creatinine Sodium pH
Medical management of kidney stones Eradication the stone Determine stone type (treat underlying cause) Prevent nephron destruction Control infection Relive obstruction (if present
Nursing management of kidney stones Relive the pain Pharmacological Opioid analgesics NSAIDS Inhibit the synthesis of prostaglandin E Reduce swelling to help facilitate passage of the stone Heat Increase fluid intake Reduces crystalloids in the urine Dilates the urine
surgical removal Only used if does not respond to traditional methods and useful to correct obstruction and damage Nephrolithotomy Incision into the kidney to remove the stone
kidney Pyelolithotomy Incision into the renal pelvis to remove the stone
kidney Ureterolithotomy Incision to remove a stone in the ureter
kidney Cystotomy Removed from the bladder
Nephrectomy removal of the kidney
Nutritional Management Calcium Stones Questionable to limit dietary intake of calcium Decrease sodium and protein Increase fluid intake Management of medications for underlying disease process High fiber diets Provides phytic acid which binds with dietary Calcium
Uric acid stones Low purine diet: Avoid shellfish, anchovies, asparagus, mushrooms and organ meats
Cystine stones Low protein diet < 60g/day Increased fluid intake
Oxalate stones Foods to increase excretion of oxylate: spinach, strawberries, rhubarb, chocolate, tea, peanuts and wheat bran
Bladder cancer incidence Age 50-70 Affects men 4:1 over women More common in Caucasians 4th leading cause of cancer in men Highest world-wide incidence Risk factors are smoking, other cancers, frequent UTI’s, and other cancers
Clinical manifestations of bladder cancer Usually arise from the base of the bladder Painless Hematuria Complications: UTI Pelvic or back pain with metastasis
Assessment/Diagnostic Findings bladder cancer Cystoscopy (Main Dx) Excretory urography Ultrasonography Bimannual examination Biopsies Staging – saline bladder washings
medical management bladder cancer Treatment is dependent on: Grade of the tumor – cellular differentiation Stage of tumor growth – the degree of invasion and metastasis Multicentricity – multiple centers of growth
Intravesical Therapy (Chemotherapy Agents are instilled into the bladder and retained for 2 hour increments. The patient’s position is changed every 2 hours to ensure contact and coating of the bladder. Typical side effects of chemotherapy are not experienced
Intravesical Therapy (Chemotherapy Urinary disruptions and incontinence are common after treatment for bladder cancer
Radical cystectomy – most preferred Bladder, prostrate and seminal vesicles and prevesical tissues in men Bladder, lower ureter, uterus, fallopian tubes, ovaries, anterior vagina and urethra in women
Transurethral resection followed by intravesical administration of bacille Calmette-Guérin (BCG) causing local inflammatory responses
Radiation Therapy Pre-operative Inoperative tumors Hydrostatic Therapy Investigational Therapy Palliative or curative Hematoporphyrin changes to a toxic medication through laser light treatment
Urinary Diversion Occurs after needed cystectomy or other neurological damage, trauma or chronic and last resort of management of incontinence
Urinary Diversion procedure Procedure is based on the degree of damage, age of the client and ability to learn post-operatively and the renal function of the client
Types of Urinary Diversions Ileal Conduit (ileal loop) Incontinent diversionary device Cutaneous Ureterostomy Continent Nephrostomy Catheter of the kidneys
Complications Wound infection Wound dehiscence Urinary leakage Urethral obstruction Hypochloremic acidosis SBO Ilius Gangrene of the stoma
Nursing Management Post-Operatively Hourly urine output > 30 mL/hr to monitor for: Dehydration Obstruction of the Ileal conduit May require a catheter and irrigation of 5-10mL of NSS
stoma care Monitor for; Infection Irritation and bleeding Encrustation and skin irritation (from alkaline urine
Monitor urine pH < 6.5 through administration of ascorbic acid
Teaching Some mucous may be present in the urine Monitor for odor of urine (could indicate infection or poor hygiene of the appliance) Encourage fluids
Changing of the appliance Early am when urineamountsdecreased Skin barrier is essential Avoid moisturizing soaps Avoid asparagus, cheese & eggs (Protein)Empty pouch when 1/3 full Clean system in 3:1 solution of water/white vinegar for 30 minutes stored after powdered w cornsta
Continent Urinary Diversions differences Post-Operatively there are more drains that need careful monitoring for drainage Cecostomy tube MUST be irrigated 2-3 times daily Indiana Pouch Kock Pouch
Patient Teaching Esteem Catheterization every 6 hours and daily irrigation
Continent Urinary Diversions- Ureterosigmoidostomy drawbacks Urinate through the rectum Have frequency/urgency
Ureterosigmoidostomy Advantages No external appliance
Ureterosigmoidostomy disadvantages Lifestyle changes from urgency/frequency Urinate though the rectum Cannot be used if the anal sphincter doesn’t function well
Ureterosigmoidostomy complications Pyelonephritis Adenocarcinoma of the sigmoid colon
Ureterosigmoidostomy nursing care Liquid diet post-operatively Antibiotics Catheter to drain the urine Tube needs irrigation Electrolyte Imbalances /Acidosis treated with low chloride diet and sodium-potassium citrate Monitor 4 s/s of UTI & drain every 2-3 hours to prevent complicat
Created by: lupde01
Popular Nursing sets

 

 



Voices

Use these flashcards to help memorize information. Look at the large card and try to recall what is on the other side. Then click the card to flip it. If you knew the answer, click the green Know box. Otherwise, click the red Don't know box.

When you've placed seven or more cards in the Don't know box, click "retry" to try those cards again.

If you've accidentally put the card in the wrong box, just click on the card to take it out of the box.

You can also use your keyboard to move the cards as follows:

If you are logged in to your account, this website will remember which cards you know and don't know so that they are in the same box the next time you log in.

When you need a break, try one of the other activities listed below the flashcards like Matching, Snowman, or Hungry Bug. Although it may feel like you're playing a game, your brain is still making more connections with the information to help you out.

To see how well you know the information, try the Quiz or Test activity.

Pass complete!
"Know" box contains:
Time elapsed:
Retries:
restart all cards