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NUR_1213 Exam 4

Oxygenation: Dysrhythmias/EKG

QuestionAnswer
Sinus Rhythm- Electrical activity of the heart initiated by the SA node. <center> occurs when the electrical impulse starts at a regular rate and rhythm in the sinus node and travels through the normal conduction pathway.</center>
Normal Sinus Rhythm (Characteristics) V&A RATE: 60-100<center> V&A RHYTHM: regular</center><center>QRS: usually normal but maybe regularly abnormal</center><center>Pwave: normal & consistant; always in front of QRS</center><center>PR INTERVAL:0.12-0.20secs</center>P:QRS ratio: 1:1
Sinus Bradycardia- Occurs when sinus node creates an impulse at a slower-than-normal rate.
Causes of Sinus Bradycardia- Lower metabolic needs (ie: sleep, athletic training, hypothyroidism), Vagal stimulation (from vomitting, suctioning, sever pain, extreme emotions), Medications (calcium channel blockers, amiodarone, beta blockers), Increased ICP, MI (esp of inferior wall)
H & T's of Sinus Bradycardia- <center> hypovolemia </center><center> hypoxia </center><center> hydrogen ions (acidosis) </center><center> hypo- or hyper- kalemia </center><center> hypoglycemia </center><center> hypothermia </center><center> _______________________ </center><center> to
Characteristics of Sinus Bradycardia... Ventricular & Atrial Rate: less than 60<center> Ventricular & Atrial Rhythm: Regular </center><center> QRS Shape & Duration: Usually normal, but may be regularly abnormal </center><center> P wave: Normal & Consistant shape; always in front of QRS </center
Only difference btwn Sinus Rhythm & Sinus Bradycardia? Ventricular & Atrial Rate
Medication of Choice in Treating Symptomatic Sinus Bradycardia? Atropine, 0.5 mg given rapidly as IV bolus q3-5mins to a max total dose of 3mg
Sinus Tachycardia- Occurs when the sinus node created an impulse at a faster then normal rate.
Causes of Sinus Tachycardia- *physiologic or pathological stress (acute bld loss, anemia, shock, hypervolemia, hypovolemia, heart failure, pain, hypermetabolic stated, fever, exercise, anxiety)<center> *Medications that stimulate the sympathetic respose (catecholamines, aminophylline
Characteristics of Sinus Tachycardia... V&A rate: >100 (but usually <120)<center> V&A rhythm: Regular </center><center> QRS shape & duration: Usually normal, but may be regularly abnormal. </center><center> P wave: Normal and consistant, always in front of QRS, but may be burried in the precced
If rapid rate of Sinus Tachycardia persists and the heart cant compensate for the decreased ventricular filling, the patient may develop? Acute pulmonary edema
Characteristics of Sinus Tachycardia... V&A rate: >100 (but usually <120)<center> V&A rhythm: Regular </center><center> QRS shape & duration: Usually normal, but may be regularly abnormal. </center><center> P wave: Normal and consistant, always in front of QRS, but may be burried in the precced
If rapid rate of Sinus Tachycardia persists and the heart cant compensate for the decreased ventricular filling, the patient may develop? Acute pulmonary edema
Sinus Tachycardia = Nonparoxysmal does not start or end suddenly
Atrial Fibrillation- causes a rapid, disorganized, and uncoordinated twitching or atrial musculature.
A Fib = Paroxysmal may be transient, starting and stopping suddenly and occuring for a very short time. <center> may also be persistant, requiring trtmnt to terminate the rhythm or to control ventricular rate. </center>
Characteristics of Afib... V&A rate: Atrial rate is 300-600, ventricular rate is usually 120-200 in untreated afib.<center> V&A rhythm: Highly irregular </center><center> QRS shape/duration: usually normal, but may be abnormal </center><center> P wave: No discrenible P waves, irreg
What treatment is indicated for afib that is hemodynamically unstable? Electrical cardioversion<center> ***************** </center><center> d/t the high risk for emobolization or atrial thrombi, cardioversion or afib that has lasted longer that 48hrs should be avoided unless the pt has recieved anticoagulants. </center>
V Tach- 3 or more PVCs in a row, occuring at a rate exceeding 100 BPM. Usually associated w/ CAD & may precede vfib.
V Tach = emergency b/c? Pt is usually (not always) unresponsive & pulseless.
V Tach Characteristics... V&A rate/rhythm: Ventricular rate = 100-200BPM. Atrial rate depends on underlying rhythm (ie-sinus rhythm);regular <center>QRS: 0.12sec or more; bizarre, abnormal shape</center><center>Pwave: difficult to detect, atrial R&R poss. indeterminable</center><c
center>PRwave: very irregular, if P waves are seen</center>P:QRS ratio: difficult to determine, but if P waves are apparent, there are usually more QRS complexs than P waves.
A patient with stable Vtach would most be treated with what choice medication? Amiodarone IV (antidysrhytmic medication of choice)
A symptomatic patient with monophasic Vtach would most likely recieve what type of treatment? Cardioversion
Torsaded de pointes- is a polymorphic Vtach preceded by a prolonged QT interval.
What should be the suspected cause of Vtach w/ an irregular rhythm? Afib
What is the action of choice for a patient w/ any type of Vtach who is unconscious & pulseless? Immediate Defibrillation!!!! (pt treated in same manner as vfib)
V Fib- rapid, disorganized ventricular rhythm that causes ineffective QUIVERING of the ventricles. (no atrial activity seen on ECG.)
Causes of Vfib? same as for Vtach; may result from untreated or unsuccessfully treated Vtach, electrical shock, Brugada syndrome.
Brugada Syndrome- (freq. of asian decent) has structurally normal heart, few or no risk factors for CAD, and family HX of sudden cardiac death.
Vfib Characteristics... Ventricular rate/rhythm: >300/min; extremely irregular, w/o specific pattern.<center>QRS: irregular, undulating waves w/o recongnizable QRS complexes</center>absence of audible heartbeat, palpable pulse and respirations.
In this arrythmia there is no coordinated cardiac activity, and cardiac arrest and death are imminent if not corrected? Vfib
Treatment of choice for Vfib? immediate bystander cardiopulmonary resuscitation (CPR), defibrillation asap, activation of emergency services.
A patient w/ Afib should be administered what medication asap after 2nd rhythm check? Vasoactive medications- epinephrine, vasopressin, or both.
A patient w/ Afib should be administerd what medication asap after 3rd rhythm check? (immediatel before of after 3rd defibrillation) Antidysthymis medications- amiodarone, lidocaine or possibly magnesium
a pt who is unconscious, who experiences cardiac arrest d/t vfib is recommended by the AHA guidelines to induce ___________? mild hypothermia for 12-24 hrs. (core body temp of 89.6-93.2*F (32-34*C))
Asystole characteristics.. absent QRS complexes confirmed by 2 diff. leads, although P waves maybe apparent for a short duration. No heartbeat, no palpable pulse and no respiration.
Asytole treatment? (same as PEA)rapid assessment to find poss. cause, 2 mins or 5 cycles of CPR, bolus of IV epinephrine is administered and repeated at 3 to 5 min intervals. 1 dose of vasopressin may be admin. for 1st or 2nd dose of epi. 1mg bolus IV atropine may be admin
Possible causes of asytole? hypoxia, acidosis, severe electrolyte imbalance, drug OD, hypovalemia, cardiac tamponade, tension pneumothorax, coronary or pulmonary thrombosis, trauma, hypothermia.
What is the normal PR interval? 0.12-0.20 seconds
PR interval- measured from begining of P wave to the begining of QRS complex and represents time needed for SA node stimulation, atrial depolarization, conduction through AV node b4 ventricular depolarization.
What is the normal QRS duration? >0.12 secs
QRS- represents ventricular depolarization
What is the normal pace maker of the heart? SA node
SA node rate? 60-100
AV node rate? 40-60
P to T represents? 1 heart beat
U wave found in patients with? hypokalemia, htn. heart disease
Horizontal = time
Vertical = voltage
How do you calculate the HR on a 6 sec. strip? # of QRS intervals (in 6 sec. strip) X10 or # of R-R intervals (in 6 sec. strip) X10
What do the small boxes represnt? (time) 0.04 secs
What do the big boxes represent? 0.20 secs (5 small boxes)
Cardiac Tamponade- pericardium fill up w/ fluid
sinus rhythm starts? narrow
v tach = 3 or more Y beats on rate >100 BPM
Carsioversion- electrical current administration in synchorony witht the patients own QRS complex to stop dysrhythmia
Defibrillation- electrical current administered to stop a dysrhythmia, not synchronized with the pt's QRS complex.
What nursing DX would be most applicable to a pt dysrhythmias? decreased cardiac output <center>anxiety related to fear of the unknown</center>Deficient knowledge about the dsyrhythmia and its treatment
Depolarization- process by which cardiac muscle cells change from a more negatively charged to a more positively charged intracellular state.
Repolarization- process by which cardiac smooth muscle cells return to a more negatively charged intracellular condition, their resting state.
difference btwn cardio version and defibrillation? cardio version- involves delivery of a "timed" electrical current to terminate a TACHYDYSHYTHMIA. Done on conscious pt<center>defib- used in emergency situations at trtmnt of choice for vfib, pulselessness Vtach. not used on conscious pts who have a pulse
What is the nurse's 1st action when told that a pt's rhythm has changed on the monitor? Check the patient
Created by: lprovoost
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