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insulin

diabetes and insulin

QuestionAnswer
Insulin Required for type 1
Perscribed for pt. with type 2 who canot control blood glucose by other means
human insulin *most widely used*cost effective*decrease likelihood of allergic reation
conventional insulin regimen for terminally ill, the frail or patients who are completely unwilling or unableavoid acute complicationsshort and intermediate acting
intensive insulin regimen *complex*maintains blood glucose*Prevents or slows the progression of long_term diabteic complications*Risk of severe hypoglycemia
rapid acting lispro
short acting regular
intermediate acting NPH or Lente
Long-acting Ultralent, Lantus
admin of insulin SQ for selfIVinhaled
what are oral agents not insulinwork to improve the mechanism in which insulin and glucose are produced and used by the body
5 oral agents 1st gen2nd genbiguanidesalpha glucosidase inhibitorsthiazolidinediones
Drug therapy oral agents: drugs that affect blood glucose levels beta adrenergic blockersthiazide loop diuretics
Acute complicationso of diabetes *hypoglycemia*diabetic ketoacidosis*hyperglycemic hyperosmolar nonketotic syndrome or coma (HHNS) or (HHS)
hypoglycemia abn. low blood glucose level (below 50-60 mg/dl)causes too much insulin or oral hypoglycemic agents, too little food and excessive physical activity
ADRENERGIC manifestations of hypoglycemia sweeting, tremors, tachycardia, palpitations, nervousness, hunger
management of hypoglycemia treatment is immediate15 g of fast acting concentrated carb: 3 or 4 glucose tabs, 4-6 ounces of juice or regular soda, 6-10 hard canides, 2-3 teaspoons of honey.retest blood glucose in 15 min. retreat if > 70mg/dl or if symptoms persis more than 10 - 15 m
EMERGENCY: if patient cannot swallow or is unconcious subQ or IM glucagon 1 mg25-50 mL 50% dextrose solution IV
diabetic ketoacidosis is caused by an absence of or inadequate amount of insulin resulting in abnormal metabolism or carg, protein, and fat.
clinical features of DKA hyperglycemiadehydrationacidosis
manifestations of DKA polyuria, polydipsia, blurred vision, weakness, headache, anorexia, abdominal, pain, nausea, vomiting, acetone breathhyperventilation w/ kussmaul resp.mental status change
treatment of DKA rehydration with IV fluidIV continuous infusion of regular insulinreverse acidosis and restoration of electrolyte blance
treatment of DKA: monitor blood glucoseEKG and electrolyte: potassiumVS lung assessments, signs of fluid overload
HHNS LACK OF INSULIN,KETOSIS IS MINIMAL OR ABSENT HYPERGLYCEMIc CAUSES OSMOTIC DIURESIS W/ LOSS OF WATER AND ELECTROLYTESHYPERMATREMIA AND INCREASED OSMOLATLITY OCCUR
MANIFESTATIONS OF HHNS HYPOTENtION, DEHYDRATION, TACHYCARDIA, NEUROLOGIC SIGNS, HIGH MORTALITY
SICK DAY RULES *take insulin or oral anitdiabetic agents as usual*test blood glucose*test urine ketones every 3-4 hours*report elevated glucose levels*if you take insulin, you may need supplemental does or regular insulin every 3-4 hours*if unable to follow meal plan, s
Created by: leah_76
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