Session 2 CMCardio6 Word Scramble
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Question | Answer |
what are the manifestations of Ischemic Heart Disease (coronary artery disease) | stable angina acute coronary syndromes CHF sudden cardiac death silent ischemia |
Due to fixed coronary lesion leads to mismatch of myocardial metabolic O2 supply and demand increases cardiac work load | stable angina |
What does Acute Coronary syndrome encompase | includes conditions compatible with MI unstable angina STEMI NSTEMI |
What happens to atherosclerotic plaque in an acute event | it ruptures and creates a thrombogenic surface within blood vessel that leads to platelet aggregation/ fibrin formation limited blood flow distal to lesion mismatch O2 supply/demand tissue ischemia (unstable angina) tissue necrosis (MI) |
What are the four grades of Acute Coronary Syndrome | class I- angina occurs only with strenous activity Class II- angina occurs with moderate activity Class III- angina occurs withe mild activity Class IV- angina occurs with any activity and may occur at rest |
What is unstable angina | rapid increase in coronary stenosis but incomplete occlusion chest pain at rest on effort w/ change from previous pattern previous stable angina becoming severe |
What are unstable angina patients at definite impending risk of | myocardial necrosis |
Apart from angina what other conditions lead to supply demand mimatches | aortic stenosis thyrotoxicosis profound anemia |
What do IHD/CAD predispose patient to | heart failure cardiac arrythmias sudden cardiac death |
Name 5 risk factors for IHD/CAD | age >65 Male>Female Smoking HTN Fam Hx DM obesity sedentary life style elevated cholesterol inflammation |
What is the most common cause of IHD/CAD and other causes | atherosclerosis spasm Arterial Stenosis Hypertrophic obstructive cardiomyopathy metabolic syndrome/ syndrome X severe HTN |
when does initial onset of atherosclerosis occur | childhood and adolescence fatty streaks with lipid filled smooth muscle cells and macrophages (foam cells) and intermittent fibrous deposition |
What happens to the atherosclerosis generally in third decade of life | fibrous plaques appear in the abdominal aorta, coronary and carotid arteries. they are firm, elevated dome shped lesions on surface that bulge into lumen |
What are atherosclerotic plaque made of | extracellular lipid with cholesterol crystals and necrotic cell debris |
What artherosclerotic plaques are usually associated with symptoms | complicated lesions of calcified fibrous plaque containing degrees of necrosis, thrombosis and ulceration |
What size vessels does atherosclerosis typically effect and how is it characterized | disease of large and medium sized muscular arteries characterized by endothelial dysfunction vascular inflammation buildup of lipids, cholesterol, calcium, and cellular debris withing intima of vessel wall |
What are the results of plaque formation | vascular remodeling acute and chronic luminal obstruction abnormalities of blood flow diminished oxygen supply to target organs |
What happens if the enothelium or fibrous cap over a atherosclerotic plaque ruptures | you get exposure of the thrombogenic contents of the core to circulating blood. This is an advanced or complicated lession and inflammatory cells localize to the plaque. T-lmphocytes release inflammatory factors |
what is the leading mechanism accepted regarding atherogenesis | response to injury where endothelial injury causes vascular inflammation and followed by fibroproliferative response |
What is the major concern of a plaque rupture | that a thrombus will develop and occlude the blood vessel |
When is anginal threshold typically lowest | More in the A.M. |
What type of angina is variable defies any pattern and is prominent at rest. | Prinzmetal Angina (variable angina) |
What might prinzmetal angina be caused by | myocardial ischemia caused by coronary spasm |
If you have angina lasting > 20min and it is severe chest discomfort accompanied by profound fatigue what does it often equal | acute myocardial infarction or acute coronary syndrome |
How long does typical angina pectoris last | 2-10 minutes |
if angina last for 15-20 minutes what type of angina is it typically representative of | unstable angina |
If the angina last for hours what might be underlying cause | acute MI pericarditis aortic dissection musculoskeletal disease herpes zoster anxiety cocaine abuse |
if chest pain is really brief less than 15 seconds is it angina | no pain points to musculoskeletal pain, hiatal hernia or functional pain |
what can be given to relieve angina pain | nitro |
If your patient is having chest pain and leaning forward helps relieve the pain more than just quiet standing or sitting what besides angina is likely causing there pain | acute pericarditis |
in angina what position is best to rest in to relieve pain lying down or standing up | standing up or quietly sitting is best lying down does not relieve angina |
what are anginal equivalents | discomfort is limited to area of secondary radiation such as lower jaw teeth neck shoulders nausea indigestion dizziness diaphoresis |
What other s/sx accompanying angina should be concerning | profuse sweating- may indicate acute MI or aortic dissection, or Pulmonary Effusion nausea and vomiting- MI SOB-MI, pneumothorax or Pulmonary effusion Palpitations- Acute MI |
What is important about angina in women, diabetics and elderly | it presents atypically. more vasospastic, microvascular angina |
When do women often report chest discomfort | at rest during sleep during mental stress |
What is dyspnea | abnormally uncomfortable awareness of breathing |
if you have sudden development of dyspnea what might that indicate | pulmonary embolism, pneumothorax, acute pulmonary edema, pneumonia, or airway obstruction |
If dyspnea develops slowly overtime what might that indicate | suggest chronic heart failure |
if you have exertional dyspnea what might that indicate | left ventricular failure, ischemic heart disease or chronic obstructive lung disease. |
if you have inspiratory dyspnea what might it be | suggest upper airway obstruction |
if you have expiratory dyspnea what might it be | lower airway obstruction |
if you have dyspnea at rest what might it be | pneumothorax, pulmonary embolism, pulmonary edema, anxiety |
if dyspnea is an anginal equivalent what may it be associated with | sensation of tightness in the chest present on exertion or emotional stress relieved by rest similar to angina in duration usually responive to or prevented by nitro |
What is orthopnea | discomfort breathing while lying down patients with left ventricular failure start sleeping with 2 or more pillows to avoid the orthopnea |
what are the three types of syncope | cardiac syncope aortic stenosis syncope neurocardiogenic syncope/ syncope due to postural hypotension |
What are palpitations | unpleasant awareness of forceful or rapid beating of the heart |
how are palpitations described | Pounding Jumping or Skipping a beat Racing Irregularity of the heart beat A "flip flopping" A "rapid fluttering" Pounding in the neck Butterflies in my chest |
If your patient has claudications ie Peripheral Vascualr disease what else do they likely have | 50% likelihood of having CAD |
What might you find on PE in angina | could have S3 or S4 xanthomas (cholesterol filled nodules) signs of CHF |
what is the most consistent ECG finding during acute ischemia | deviation of ST segment from current of injury mechanism |
what is the management for angina | risk factor modification asa daily NTG prn long actin nitrates BB, CCB PTCA(PCI) CABG |
what is the most common cause of MI | atherosclertotic palque ruptures stimulating thrombus formation |
What is the clinical findings for MI | Severe anginal type pains, usually lasting >30 minutes, NOT relieved by NTG or rest SOB Anxiety Diaphoresis N/V Syncope Atypical & silent presentations |
Created by:
smaxsmith
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