Autonomic Innervation, Hormones, Drugs
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| Question |
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| Answer |
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| Where does the ANS innervate for the control of contraction? |
exerted on SA and/or AV Nodes |
| Which division of the ANS is normally dominant? |
Parasympathetic |
| What Neurotransmitter & receptor type does the Parasympathetic use? |
Acetylcholine - Cholinergic receptors |
| What effect does Acetylcholine have on RP? |
Hyperpolarize |
| What is the function of the Parasympathetic? |
REDUCES HR and Contractility |
| What is the significance of the Parasympathetic on the heart? |
HYPERPOLARIZES Cardiac RP via |
| Where do Sympathetic impulses come from? |
Thoracic Nerve Branches |
| Where do Parasympathetic impulses come from? |
Vagus Nerve |
| Where does the Neurotransmitter Epinephrine come from? |
Adrenal Glands |
| What are the 2 receptors that accept Epinephrine? |
ß-1 and ß2 |
| What is the result of Epinephrine binding to a ß-1 receptor? |
DEPOLARIZATION --> INCREASE Rate/Force of Contraction |
| What is the result of Epinephrine binding to a ß-2 receptor? |
HYPERPOLARIZATION --> Vasodialation |
| What 3 factors influence HR? |
Drugs, Hormones, ANS |
| What are the 2 types of Drugs which effect the control of contraction? |
Cholenergic blockers & ß-blockers |
| What type of blocker is Atropine, and how does it work? |
Cholenergic - blocks Acetylcholine --> blocks Parasympathetic --> |
| How do ß-blockers work? |
blocks ß-1 --> cancel Noerepinephrine --> DECREASE HR/BP |
| What is the Neurotransmitter used by the Sympathetic NS? |
Norepinephrine |
| How does Norepinephrine effect the Heart? |
DEPOLARIZES Cardiac RP via ^ Na+/Ca++ Permeability |
| What is the function of the Sympathetic division of the ANS? |
INCREASES HR and Contractility |
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