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Cardiovascular disorders

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Question
Answer
cardiovascular system   artiers, veins capillaries  
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Arteries   carry oxygenated blood from heart to body, muscular and elastic in nature-accomodate pressure increases during heart contraction, mainaint unidirection blood flow duint heart relaxation  
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veins   composed of small layer of muscle, blood flow back to hear it supported by skeletal muscle contraction, valves present to prevent backflow of blood  
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Capillaries   connect artiers to veins through networks fo tiny vessels-capillary beds, exchangfe of nutrients, gases horomones  
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Blood flow   left and right sides of heart work together, both bentricles contract(systole), releax(diastole), heart pumps 5-6L of blood per min, contraction ciculates blood to every cell in body  
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Diasotle   relaxation-blood flows into and through atria to ventricles, as atrial pressure increases, blood flow forced down into ventricles  
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systole   contraction-during ventricular contraction-tricuspid valve closess-blood travels to pulmonary artery, mitral valve closes-blood travels to aortic atery  
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Stroke Volume   amount of blood pumped during ventricular contraction  
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Cardiac output   stroke volume* heart rate  
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Heart internal structures   coronary artiers, intrinsic electical conduction system  
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coronary arteries   branch from base of aorta to supply entire myocaridum  
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intrinsic electrical conduction system   SA node depolarizes-spread electicity though pathway in atria, depolarization reaches the AV node-travels to ventricular walls-contraction by wya of AV bundle, bundle branches, pukinji fibers  
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Responses and adapations to exercise   exercising muscls require more o2, both components of cardiac output are increased, vascular resistence decreases,-vasodilation in skeletal muscle, systolic pressure can increase to 250mmHg, diastolic pressure remains constant  
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exercising muscles require more o2   heart rate and respiration rate both increase significantly  
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both components of cardia output are increased   stroke volume and heart rate  
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vascular resistance decreases-basodilation in skeletal uscles   bp does not decrease b.c of increased vardiac output  
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systolic pressure can increase to 250mmhg   high systolic pressure is necessary to maintain blood flow to vessels being occluded by intense muscle contraction  
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Long term responses and adaptations to exercise   increase CO(increase in SV), cardiac hypertrophy, decreased in resting heart rate, little change in BP  
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Once exercise ceases what happens?   hr should decrease by atleast 12 bpm after the first min(mor fit, quicker it falls), BP should drop 8-12 mmHg/MET level  
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AHA minimal components   capable of identifying up to 50% of atrisk ath, follow up testing should be done for any suspected abnormalities-ECG echocardiogram, blood tests, CMR exercise testing  
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Echocardiogram   ultrasound of the heart  
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Blood tests   sickle cell trait/diseas, mycardial infarction  
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CMR   Cardiac magnetic resonance imaging  
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ECG   electrocardiogram-assesses the hearts electricle system, relitively inexpensive test, assocate w. false positives  
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Pwave   atrial depolarization  
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QRS segment   ventricular depolarization  
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Twave   ventricular repolarization  
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diagnostic testing?   some controversy as to whether ppe should include ECGs, echocardiograms, or blood testing, ATs should discuss the use of these CV screening tests w/ their team physicians, decision to include tests in standare PPE is both a philosophical and finacial one  
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