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Pharm10

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
-zosin   alpha adrenergic blocker  
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-lol   beta adrenergic blocker  
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-zepam, -zolam   BZO  
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-pril   ACEI  
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-sartan   angiotensin II receptor blocker  
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-dipine   Ca channel blocker  
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-statin   LDL-lowering HMG-CoA reductase inhibitor  
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-sone, -olone   corticosteroid  
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-parin   anticoagulant  
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-prazole   PPI  
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-terol, -phylline   bronchodilator  
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-semide   loop diuretic  
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How does alpha-2 stimulation differ from other sympathetic receptor sites?   Can inhibit release of epinephrine or NE from presynaptic neuron  
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Adrenergics, sympathomimetics, adrenergic agonists mimic the effects of?   NE  
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Sympatholytics, adrenergic blockers, and adrenergic antagonists block the effects of   NE  
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Cholinergics, parasympathomimetics, cholinergic agonists, cholinomimmetics mimic the effects of   Ach  
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Anticholinergics, parasympatholytics, and cholinergic antagonists block the effects of   Ach  
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Sx of sulfite sensitivity   Dizziness, loss of consciousness, hypotension, syncope, clammy flushed skin, pruritus, cyanosis, bronchospasm, wheezing, SOB, laryngeal edema, respiratory arrest Epinephrine, NE, phenylephrine, methoxamine, dopamine, dobutamine, metaraminol, fenoldopam  
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Pt edu for pts receiving clonidine (Catapres)   Never stop abruptly- can cause rebound HTN  
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How does dopamine (Intropin) affect renal hemodynamics?   High doses (bind alpha-1 receptors) cause vasoconstriction in kidneys, can cause renal failure. Low doses (binds dopaminergic receptors) causes increased GFR, renal blood flow, and sodium excretion (overall, increased renal output)  
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Why is dopamine (Intropin) and fenoldopam (Corlopam) only admined in acute care envts?   Need to closely monitor BP, CO, urine output, pulmonary wedge pressure b/c can cause severe dysrhythmias and vasoconstriction (dopamine) or acute cerebral infarction or hemorrhage (fenoldopam)  
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AE of prazosin (Minipress)   1st dose syncope  
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Why are beta blockers used cautiously in pts with resp disorders?   Can cause development of obstructive pulmonary disorders  
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Cholinergic crisis   overstimulation at nm junction due to too much Ach, give atropine  
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Myasthenic crisis   lack of Ach due to undermedication, give more prostigmin can cause inability to speak, breathe, or swallow in pts with myasthenia gravis  
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Common AEs of atropine   blurred vision, urinary retention, constipation, dry mouth  
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Location of beta 2 receptors   lungs  
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location of beta 1 receptors   heart, kidneys  
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location of alpha 1 receptors   prostate, peripheral vasculature  
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location of alpha 2 receptors   terminal end of presynaptic neuron  
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propranolol (Inderal) effects on blood glucose   None, but inhibits perception of hypoglycemic sx  
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Body SA   sqrt [weight (kg) x height (cm)]/3600  
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-triptans   serotonin agonists  
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LFTs   bilirubin, AST, ALT, alk phosphatase, PTT, aPTT  
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renal function tests   BUN, serum creatinine, creatinine clearance  
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hematopoietic function tests   CBC- Hgb, Hct, WBC, platelets  
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6 rights of drug admin   Right: patient, time, drug, dose, route, documentation  
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IM sites   deltoid, rectus femoris, vastus lateralis, dorsogluteal, ventrogluteal  
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half-life   time required to remove 50% of the blood [drug]  
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steady state   4-5 half lives, admin rate=excretion rate  
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