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Blood #6 - blood disorders

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anemia   not enough RBC  
hemorrhagic   rapid blood loss  
hemolytic   rupture of RBC due to infections or mis-match blood during transfusion  
aplastic   distruction of bone marrow cells by drugs, toxins, radiation  
iron deficiency   can result from hemorrhage or low iron intake  
athletes anemia   rapid expansion of blood volumn therefore low cell count  
pernicious anemia   lack of intrinsic factor leads to deficiency of vit B-12  
thalassemia   absence of globulin chains or depressed synthesis of globulin chains, which cause RBC's to be thin  
sickle-cell   abnormal hemoglobin affects RBC shape  
porphyria   warewolves, vampires, inherited, lack of heme, OR exzyme for heme production lacking OR sunlite aggravates porphyrins  
polycythemia   excess RBC's  
polycythemia vera   excessive RBC production due to bone marrow cancer  
secondary polycythemia   overproduction due to RBC increase/decrease in 0 2  
blood doping   artifically induced. take out RBCs and put them back in just prior to sporting event  
leukopenia   low WBC count induced by drugs (ie anticancer drugs)  
leukemia   blood cell cancer. overproduction  
epstein-barr   infectious mono; cantagious viral disease; excessive #' of agranulocytes  
thrombocytopenia   platelet deficiency causing spontaneous bleeding. fewer than 50M platets per cc3.  
hemophilia AKA bleeders disease   TYPE A: lack of factor VIII (AHF-antihemolytic factor) TYPE B: lack of factor IX (christmas factor) both forms are sex linked traits  
Thrombus   clot formation thta occurs when clots form in unbroken vessels  
embolus (plural+emboli)   when a clot breaks loose and travels  


   


 

 

 

 

 

 
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