Adv. Health Ass. test 1
Quiz yourself by thinking what should be in
each of the black spaces below before clicking
on it to display the answer.
Help!
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| types of anesthesia | general, regional, monitored
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| what does ASA stand for | American Society of Anesthesiologist
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| what does ASA stand for | American Society of Anesthesiologist
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| ASA guidelines is | formulated health categories as guidelines for anesthesia administration
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| ASA III is | pt with severe systemic disease (HTN,DM)
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| ASA V | pt with high morbidity
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| ASA I | normal, healthy pt
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| ASA IV | pt with severe systemic disease that is a threat to life
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| ASA II | pt with mild systemic disease
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| ASA VI | brain death
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| what valves close during S1? | tricuspid and mitral valve
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| during S2 which valves close? | aortic and pulmonary
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| what is the order of valves closure? | MV, TV, AV, PV
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| systole takes twice as long as diastole? T or F | False;diastole is twice as long
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| when does the longer pause occure b/n S1 & S2? | b/n S2 and S1
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| in a healthy adult a splitting of S2 can be heard during? | deep inspiration
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| when is the pulse palpable (S1,S2) | S1
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| what is the location to hear the aortic valve? | 2nd ICS Rt. sternal border
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| where is S2 the loudest? | 2nd ICS, Rt. sternal border
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| mitral stenosis is what type of murmur | holodiastolic murmur
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| where is S1 heard the loudest? | 5th ICS
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| where is the location for auscultating the tricuspid valve | Lt. lower sternal border
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| Aortic stenosis is what kind of murmur? | holosystolic murmur
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| intensity of the murmur is graded according to? | Levine Scale
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| Level IV murmur? | medium intensity with a palpable thrill
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| ___ is a common high-volume state were physiologic flow murmurs are often heard. | Pregnancy
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| ___ & ____ can cause high-flow situations when the murmur is not pathologic itself, but indicates an underlying disease process. | Anemia & thyrotoxicosis
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| what pt. population can have a innocent murmur? | Children
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| Level II murmur | low intensity, but usually audible by all listeners
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| Level V murmurs? | loud intensity with a palpable thrill, audible even with the stethoscope placed on the chest with the edge of the diaphragm.
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| Level I murmurs | lowest intensity, difficult to hear even by expert listeners
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| Level III murmurs | medium intensity, easy to hear even by inexperienced listeners, but without a palpable thrill.
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| what are some murmur symptoms? | syncope, CP, palpitations, SOB, or paroxysmal nocturnal dyspnea
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| murmurs tend to change intensity with positional maneuvers, becoming louder with standing and quieter with squatting T or F | louder=squatting
quieter=standing
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| a valsalva maneuver will increase the intensity of the murmur? T or F | false;decrease
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| what is a common murmur in children and the location? | venous hum;Rt. clavicle, radiates to the neck.
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| what is a venous hum? | a continuous murmur which does not change from systole to diastole, common in children
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| how can venous hum be obliterated? | by brief digital pressure on the ipsilateral internal jugular vein.
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| atrial septal defect; what prominent sound is heard and location? | mid-systolic flow murmur, wide "fixed" split S2/pulmonic area
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| ventricular septal defect is best heard where and what kind of murmur? | tricuspid (lower Lt. sternal border) with radiation to the Rt. lower sternal border/holosystolic murmur
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| whats the characteristic sound of mitral valve prolapse | mid-systolic click
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| immediately after the click, a brief ____-____ murmur is heard, usually best at the apex | crescendo-decrescendo murmur
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| MR is best heard at the apex with radiation to the ___ | axilla
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| quality usually describled as blowing, often associated with an S3 | MR
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| causes of AR | 66% rheumatic heart disease, congenital or associated with syphilis infection, Marfan syndrome, or valvular deterioration, infective endocarditis
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| an early mid-systolic flow murmur is freq. audible over the rt. upper sternal border with radiation to the neck | AR
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| and austion flint is what type of murmur | AR
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| this murmur can be heard radiating into the neck or the back, has a crescendo-descrescendo shape, and a harsh quality | PS
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| hear at the second ICS along the left sternal border | PS
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| best heard with the pt in the lt. lateral decubitus position | MS
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| all cases are caused by rheumatic fever in origin or congenital 2:1 F:M | MA
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| AS is best heard | over right second ICS, mid-sys eject murmur
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| what are the aortic stenosis complications ASC | Angina, Syncope, CHF
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| with AS an early peaking murmur is usually associated | with a less stenotic valve
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| with AS a more severe degree of stenosis can be differentiated by | late peaking murmur
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| murmurs tend to be located b/n the ___ & ___ , have minimal radiation, occur during early to mid-syst, have a crescendo-decrescendo shape, and a vibratroy quality. | apex & left lower sternal border
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| this is the main substrate for energy production; oxidative phosphorylation | glucose
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| increased glucose in the bld..increased ___ ___ | brain metabolism
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| what is used interchangeably with CMRO2 | CMRglucose
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| brain receibes __% of cardiac output | 15
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| coupling mechanism belong with | CBF and CMRO2
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| increase CBF in one region= increase in ____ same region | CMRO2
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| increase in CO2 will | vasodilate and increase CBF
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| what directly and indirectly effects vasodilation effects | indirectly CO2
directly H
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| what does luxury mean | increase in CBF; luxuay flow to normal brain
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| what does stea mean | decrease in CBF; steal away flow from injured areas(requires more O2)
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| what is the robinhood or inverse steal effect | ischemic/comprised areas are already dilated maximally, bld would shunt to the maximally dilated atrterioles
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| what is beneficial to the brain if a clots occurs in certain areas in the circle of willis? | there are communication arteries that could provide bld to the occluded side
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| autoregulation of CBF normotensive=____ mmHg MAP | 50-150
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| autoregulation of CBF hypertensive=___mmHg MAP | 70-170
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| <50mmHg MAP= | mild s/s of cerebral ischemia
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| disruption of BBB...cerebral edema for autoregulation of CBF | >170
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| what are the 4 varaibles on the autoregulation chart and what are the x and y axis | PaO2, PaCO2, CPP, ICP
x=pressure y=CBF
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| most electrolytes values in the CSF>ECF T or F | false; greater in CSF
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| what is choroid plexus? | (highly vascular folds of pia) of the cerebral ventricles produces CSF
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| CSF is reabsorbed into the venous system of the brain from what? | arachnoid villi
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| normal CSF volume | 100 to 150mls
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| CSF is formed and reabsorbed at a rate of | 0.3 to 0.4ml/min
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| CSF is replaced | 3 to 4x/day
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| causes of CSF BBB disruptions | Acute HTN, osmotic shock, diseases, tumor, trauma, irradiation, ischemia
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| cranium content volumes | Bld 12%(CBF)
Brain 80% (BTV)
CSF 8%
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| normal ICP | <10 mmHg
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| first and major component to reduce is with ICP | MAP
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| what are the two major effects and causes of increased ICP | brain ischemia
brain herniation
increased CSF:blockage of CSF absorpt.,formation
increased CBF:vasodilation hematoma
increased TBV: tumor or edema
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| Which have coupling effects inhalations or IV meds | IV meds
increase in both CBF and CMRO2
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| what are the EEG waves | Delta, Theta, Alpha, Beta
D:deep S/T:sleep A:awake B:concentrating
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| what is the range for BIS monitoring for general anesthesia and lt./mod. sedation? | 40-60;70-90
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| these monitor the integrity of specific sensory and motor pathways | EP evoked potentials
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| what are EP measured by | ht and wt
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| IV anesthetics and volatiles __ amplitude, and __ latency of responses. | decrease, increase
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| which are very, somewhat, and barely sensitive somatosensory EP, brainstem auditory EP, visual EP | VEP:very, SSEP:somewhat, BAEP:barely
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| what are the indications for MEP motor EP | intramedullary tumors, scoliosis surgery, cerebral tumors
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| what are the cautions of MEP | h/o seizures, poss. skull fx, implanted metallic devices, pacemakers, and CVP or PAC's
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| what are the concerns of MEP | repetitive electrical or magnetic stimulation can induce epileptic activity, neural damage and cognitive or memory dysfx
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| which cranial nerves can be preserved for motor components? and the indications | V, VII, IX, X, XI, XII
post. fossa and lower brainstem procedures
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| what is used for clinical imaging of intracranial vasculature | TCD;transcranial doppler u/s
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| transcranial oximetry uses and indications | infrared spectroscopy (NIRS); CEA, head injury, SAH clip
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| O2 sat of jugular venous is assess by | jugular bulb venous oximetry
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| brain tissue oxygenation= | PO2, PCO2, pH, and temp
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| what are the 3 main principles of radiation and which is most important? | time, distance, shielding; distance
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| what are examples of neuroradiology | CT, MRI, PET, cerebral angiography
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| generalized pre-op eval. of neurosurgery | overall medical condition, length of procedure, positioning, institutional special techniques: hyperventilation, cerebral dehydration, deliberate hypoTN
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| what are some s/s of intracranial HTN | H/A, N/V, papilledema, unilateral papillary dilation, and oculomotor or abducens palsy.
Adv: depress. LOC, irreg. RR
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| causes of supratentorial ICTumors | meningiomas, gliomas, metastatic lesions
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| what kind of posturing is noted with supratentorial | decorticate
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| what must be controlled prior to cranium opening for optimal operating conditions | ICP
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| what are the prerequisites for awake craniotomy and meds | cooperative pt, able to participate in neurocognitive testing, uncomplicated airway, candidate for GA
Propofol, dexmedetomidine, versed, fentanyl, remifentanyl
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| what does the infratentorial portio consist of | medulla, pons, cerebellun, major motor and sensory pathways, primary resp. and CV centers, and lower cranial nerve nuclei
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| what posturing is seen with infratentroial | decerebrate
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| what is one of the major complications with infratentorial intracraninal surgery | venous air embolism
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| what are the advantages of sitting position surgery | excellent surgical eposure, facilitates venous and CSF drainage, better ventilation, easy access to chest, airway, ETT, and extrem., reduced facial and conjunctival edema
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| what are causes and incidence of VAE | operative field is elevated 5 cm or more above the heart's right atrium and 40-45% operated on sitting
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| from mot sensitive to least sensitive monitors | TEE>precordial dopples>PAC>capnography(ETCO2).Mass spectrometry(ETN2)
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| VAE interventions | notify surgeon immed, surgical field flood saline and packed, bone edges waxed, N2O off, Fi02 100%, neck viens compress.=↑jugular v.p., aspirate air from TLC, ↓head to heart level, avoid PEEP or valsalva, postion horiz. if fail lt.lateral fail supine CPR
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| preop eval for pituitary tumors | assoc. med. do(HTN, CHF, obese, ↓K, metabolic alkalosis, acromegaly
2.airway condition:hoarseness, dyspnea,inspir. stridor=xray
3.approach?transphenoidal needs nasal cx to guide ax tx
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| what kind of tube is used with pituitary turmors | (RAE) Ring-Adair-Elwyn
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| what are some considerations with pituitary tumors | lateral wall:venous structures(hemorr), internal carotid artery(spasm or thrombotic occl), and CN II, IV, V and VI(weakness 2ndary streching)
front pit. stalk:optic nerves n tracts, optic chiasm(visual complicat.)
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| more considerations with pit. tumors | if visual acuity is same or improved proceed with extubate. Awake and following commands prior to extubate 2ndary to nasal packing, pt will need to mouth breathe
postop:steroid coverage, strict I&O, airway status, freq neuro checks.
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| incidences of ICA | occurs in 8 to 10 per 10,000
peak is 50's and 60's
women>men
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| IA with SAH affects | 27,000 yearly
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| risk factors for aneurysm rupture | smoking, HTN, ETOH, cocaine and amphetamine abuse, BC, plasma chl>6.3, genetic, familial(first-degree relatives)
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| some s/s of SAH | sudden severe H/A, stiff neck, photphobia, N/A, transient loss of LOC, HTN, dysrhymias
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| what effects 70% of pts with SAH | cerebral vasopasm
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| IA complications | rebleeding and cerebral vasopasm, IC HTN, acute obstructive hydrocephalus, hyponatremia/vol., seizures
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| what is triple "H" therapy | treat with hypervolemia, HYN, Hemodilution
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| IA surgical intervention endovascular txment:occ. of IA with insertion of | guglielmi detachable coils (GDC), microballoons, stents, liquid embolic agents, bioactive coils
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| goal for IA anesthesia | avoid aneurysm rupture, maintain CPP and provide a slack brain. discuss BP parameters prior to clipping
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| prior to aneurism clipping | replace NPO deficits, bld available, thiopental bolus, temp. clipping should not exceed 10 min., recirculation to be established and another thiopental bolus prior to reclipping
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| emergence goal after aneurysm clipping | avoid coughing, straining, hypercarbia, HTN
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| this consist of a tangle of congenitally malformed bld vessels that forms and abnormal communication b/n the arterial and venous syst. | AV malformation
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| management of AVMs | surgical excision, embolization, stereotactic radiosurgery, combination of above, leave it alone
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| leading causes of traumatic brain injury | MVA, violence and falls
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| intubate if GCS is | 8 or less
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| Emergency Therapy: (2) things | ABC's MILS
manual in line stabilization
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| what type of cervical spine xrays are recommended | anteroposterior and odontoid views (misses 7%)
lateral misses 20% of fx
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| cushing triad with head injuries | reflex arterial HTN, bradycardia, irregular respirations
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| anesthesia concerns with head injures | fiberoptic intubation or AII RSI, ketamine, Sch, server IC.HTN
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| cause of CVD | emboli air, fibrin or ca;ischemia;severe sustained hypoTN..IC thrombosis/infart;HTN..hemorrhagic stroke/disrput of BBB
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| anesthesia concerns with CVD | regional vs. gerneral, rightward shift of autoregulation curve, no rely on pressor=MI, avoid Sch in recent stroke pts, muscle wasting, hyperkalemia, paralysed can result to overdose(resistance to NMB)
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| classification of partial (focal) seizures | i.simple, ii.complex, iii.2ndary generalized tonic-clonic
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| classification of generalized seizures | i.absence(petit mal) ii.myoclonic iii.clonic iv.tonic v.tonic-clonic vi.atonic vii.unclassified
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| mechanisms of seizures do | loss of inhib. GABA act.,enhanced release of excitatory amino acids (glutamine), enhanced neurofiring
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| interventions during seizures | maintain an open airway with O2, IV bolus thiopental, phenytoin or benzo
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| avoid potentially epieptogenic drugs | ketamine in small doses, lg doses of atracurium, cisatracurium, meperidine
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| a pt that takes seizure meds you need to increase MAC b/c | hepatic microsomal enzyme induction
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| degenerative and demyelinating diseases | parkinson, alzheimers, MS, amyotrophic lateral sclerosis, G-B syndrome, automonic dys./dysautonomia, syringomyelia
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| intaoperative phase consist of | induction, maintenance, and emergence
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| anesthesia and elective operations should not procedd until the pt's medical condition has been | optimized
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| disease state already present or hospital acquired | morbidity
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| 2 majoy elements of preop phase | gathering info, formulation an anesthetic plan of care
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| what does the gathering info consists of | reviewof pertinent current and past med records, interview, phy. exam
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| what is the communication of the anesth. plan | informed consent
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| unlawful threat of actions by a perosn intended to inflict, my force, corporal injury | assualt
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| battery | unlawful constraint or phy. violence inflicted on a person without his consent, administation of anesthetic to an unwilling person
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| peds that are at risk for developing hypoxia intra and post op | upper resp infection
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| cross sensitives allergies | beta lactam anx, sulfa, amides
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| disadv. of ASA | imprecise, subjective, higher ASA only roughly predicts anesthetic risk
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| what is a red flag with airway assessment | old trach scar
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| medical conditions assoc. with potential airway diff. | soft tissure enlargement=obstructive sleep apnea, obestiy, epiglotittis, down's) restrictive oral opening=RA, facial trauma,receeded mandible Cervial spine abd=prev. spine fusion, ankylosing spondy. Active airway infect.=ped for T&A
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| test for airway assess. | exam oropharynx(mallampati test), thyro-mental distance, temporo-mandibular jt., neck ROM
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| class I airway | soft palate, fauces, uvula, ant. and post. tonsillar pillars
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| class III airway | soft palate, base of uvula
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| class II airway | soft palate, fauces, and uvula
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| class IV airway | soft palate only
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| mental space/thyro-mental distance | while pt in stiff posit.
normal is 6.5cm or 3-4 fingerbreathes
< 6 difficult
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| TMJ test | open mouth as wide as possible more than 2 fingerbreathes
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| Neck ROM | range of motion, ability to attain sniff position
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| no solid foods, unrestricted clear liquids, pre-op meds w/ sips | 8,2,1-2hrs
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| ped npo status | clear=2hrs
breast milk=4hrs
formula milk/solid food=6hrs
increased risk for aspiration >25cc
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| high risk for aspiration | GI ob, h/o GERD, DM, recent solid food intake, abd distent, depressed consc., up/low GIB,airway trauma, emerg. surg., N/A
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