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Stack #240092

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Question
Answer
Stroke is an   acute, local reduction in perfusion of the brain caused by occlusion,stenosis, or rupture of a vessel supplying blood to the brain  
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Types of stroke are   ischemic (accounts for 83%) or hemorrhagic  
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Ischemic there are 3 types   large artery atherosclerotic(large vessel occlusive disease), Embolism (cardioembolism), Lacunar stroke (small vessel occlusive disease)  
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Large artery atherosclerotic stroke   most often the MCA, 1/2 of the vessel is occluded by atherosclerotic plaque, & the other 1/2 by thrombus, high mortality rate  
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Embolism stroke   embolism that traveled from the heart to the brain & produced an obstruction in the brain. Also occurs due to cardiac structure change i.e. rhythm disturbance a-fib, endocarditis, valvular heart disease, AMI, CA, traumaa treated w/ coagulants  
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Lacunar stroke   seen in patients who smoke/HTN/DM  
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IC hemorrhage   damaged vessels in brain rupture, blood infiltrates parenchyma, sudden increase in pressure can damage brain and lead to ischemia distal to bleed  
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Risk factore of IC hemorrhage   uncontrolled HTN, bleeding disease, use of anticoagulants, excessive ETOH consumption & drugs  
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Subarachnoid hemorrhage   a blood vessel ruptures & blood leaks into the membrane surrounding the brain  
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Subarachnoid hemorrhage caused by   cerebral aneurysm, AVM, trauma other  
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Aneurysm   a weak area in blood vessel that enlarges out of the vessel usually in the circle of wills at the bifurcations most common & easiest to treat berry or saccular aneurysm  
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AVM   masses of abnormal vessels which grow in the brain, unruptured AVM can cause HA, sometimes the only presenting sign  
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2 Zones of injury   2 within the cerebrovascular bed core ishemic zone (NOT VIABLE LONG) penumbra (VIABLE FOR SEVERAL HOURS)  
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Intracranial aneurysm rupture results in   accumulation of blood in the subarachnoid space, clogging the arachnoid villi leading to hydrocephalous, risk for vasospasms, preventing vasospasming is vital  
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MCA stroke symptoms   loss of movement & sensation on contralateral side  
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PCA stroke symptoms   CNIII palsy, weakness & ataxia, coma, unreactive pupils & decerebrate rigidity  
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Basilary artery stroke symptoms   dizziness, lightheadedness  
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If stroke symptoms improve quickly most likely   ischemic stroke  
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Differentiate stroke from stroke mimic ie.e   seizures, systemic infections, brain tumors, toxic metabolic conditions low NA, hypo/hyperglycemia, high Ca  
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Stroke diagnosed via   noncontrast CT/MRI, blood glucose, e-, 12 lead ECG, cardiac biomarkers, CBC, PT/INR, aPTT, O2 sa  
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For candidates for treatment with tpa goal   scan completed w/i 25 minutes & interpretation w/i 20 min  
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For administration of thrombolytic therapy patient must   18 years, apparent neurological deficits consistent w/ ischemic stroke, onset of symptoms was < 3 hours before the beginnin of the infusion, Ct scan - for hemorrhage  
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BP control indicated in certain groups   thrombolytic therapy if elevates post therapy  
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Temperature control   temperatures have a poor neurological outcome in acute ischemic stroke patient  
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glucose monitoring   promotely correct hypo/hyperglycemia to prevent brain injury  
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Thrombectomy   removal of thrombus via the femoral artery up to the cerebral circulation can be done w/i 8 hours of onset of symptoms  
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Created by: tauvia2003