CardioPulmonary Physiology - Units 6-7 SPC
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| Location and shape of Kidneys | Bean shaped. Behind peritoneal cavity, below diaphragm. Cephaled poles = T12, and Caudal poles = L3...RETROPERITONEAL
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| Anatomy of the Kidney | Adrenal glands- hormones, Renal artery and vein, Ureters- urine to bladder
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| Components of the Kidney | Cortex- DARK OUTER, Medulla- PALE INNER, Renal Pyramids- 8-12 converge in to the Papillary Ducts to Major and Minor Calyces make up the Renal pelvis
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| Blood Vessles of Kidney | Renal Art, Interlobar Art, Arcuate Art, Interlobular Art, Afferent Arterioles, Glomerulus, Efferent, Peritubular Cap, Interlobular Vein, Arcuate V, Interlobar, V, Renal V
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| Nepheron | functional unit of kidney. analgous to acinus
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| Glomerulus | network of up to 50 parallel capillaries branch from afferent art.
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| Bowman's Capsule | C- shaped expanded end of renal tubule holds glomerulus. Function = FILTER
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| Nephron Components | Glomerulus, Bowman's Cap, Proximal tubule, Loop of Henle, Distal tubule, Collecting duct
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| 2 Capillary Beds of Nephron | Glomerular, Peritubular
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| Function of Glomerulus | Ultrafilter of Blood. mean P 55
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| Function of Peritubular Capillary | Tubular secretion, Tubular reabsorption
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| 3 Processes of the Nephron | Glomerular Filtration, Tubular Secretion, Tubular Reabsorption
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| Normal Glomerular Fitration Rate (GFR) | 125ml/minute
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| Urine Output | 60ml/hour or 1ml/minute
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| Glomerular Filtraion in relation to Pressure | Glom BP 55mmHg, BowCap fluid P -15, Osmotic P -30 = NET FILTRATION PRESSURE 10mmHg
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| Glomerular Blood Vessels | AFFERENT ART- constriction = Decrease GFR, dilate = increase GFR...EFFERENT ART- constriction = increase GFR, dilate = decrease GFR
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| Reabsorption of Renal tubules | Out of Tubules and into Peritubular Cap...Glucose, Sodium, Calcium, Amino Acids
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| Secretion of Renal Tubules | Out of Peritubular Cap in to Tubules....Creatinine
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| Water Transport of Tubular System | Osmosis- 80% H2O reasbsorption occurs in Proximal Tubule via Osmosis. Glucose, Sodium and Chloride have strong influence
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| Transport Maximum | MAX rate for a substance that is SECRETED or REABSORBED via active transport (TM)
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| TM of Glucose | 320mg/min
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| TM of Creatinine | 16mg/min
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| Tubular secretion of urea BUN Normal | Plasma BUN 8-18mg/dL
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| Tubular Secretion of Creatinine Normal | Plasma Creatinine 0.6-1.2mg/dL
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| AnitDiuretic Hormone (ADH) | Secreted by Posterior Pituitary Gland, Influenced by Serum Osmolarity, Increase Osmol. triggers ADH release which DECREASES Urine output= WATER RETENTION
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| Total Body WATER | Males-60%, Females 50%, Newborn-75%
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| MILLIMOLE (mmol) | 1/1000 of a mole, molecular or atomic weight in milligrams
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| MILLIEQUIVALENT (mEq)IONIC Charge | 1/1000 of Equivalent. Ionized substances. Electrolytes, Cations, anions
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| MILLIOSMOLE (mOsm) | 1/1000 of osmole. How much solute you have in plasma. Glucose and Protein exert great osmotic influence
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| Major Intracellular Cations and concentration | K-150mEq/L, Mg-25mEq/L, Na-15mEq/L
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| Major Intracellular Anions and conc. | HPO4(Phosphate)-100mEq/L, Pr(Proteinate)-60, SO4(Sulfate)-20
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| Major Plasma Cations and conc. | Na-140, K-5, Ca-5
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| Major Plamsa Anions and conc. | Cl-105, HCO3-24
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| Anions Gap | to see if metabolic imbalance. Sub HCO3 and Cl from Na and K. Normal = 10-20mEq/L
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| Clinical significance of Anion Gap | if >22= METABOLIC ACIDOSIS
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| Normal Serum Osmolarity | 275-300 mOsm/L
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| What determines Serum Osmolarity | Sodium, Glucose and BUN
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| Loop Diuretics (LASIX)FAST AND POWERFUL | inhibit reabsorption of Sodium, Potassium, and Chloride in Henle.
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| Thiazide Diuretics SLOWER | Inhibit tubular reabsorption of Sodium, Potassium, and Chloride in distal tube. Bicarb is reabsorbed = METABOLIC ALKALOSIS
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| Osmotic Diuretics (MANNITOL) | PULL a lot of fluid. Large molecular substance pass into tubules thru glomerular membrane and are not reabsorbed
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| ALDOSTERONE | adrenal hormone- Increases Sodium reabsorption and Potassium secretion in response to Hyponatremia, Hyperkalemia, Hypovolemia, Decreased CO
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| Phosphate Buffer FAST, NOT LONG LASTING | to prevent acidosis, gets rid of extra H ions by forming a salt
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| Ammonia Buffer SLOWER, LONG LASTING | gets rid of extra H ions by forming a salt, synthesized in Renal tubules, effective for long term acidosis
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| Cause of Resp ACIDOSIS | Central nervous system depression, anesthesia, sedative drugs, narcotic analgesics, barbituates, Restricive disorders like obesity and kyphoscoliosis, COPD
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| Causes of Resp ALKALOSIS | Anxiety, Stimulant drugs, Pain, Stim of J receptor=rapid shallow breathing, Pain, Pulm. Vascvular disease, Athsma
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| Causes of Met. Acidosis | Loss of Base/Bicarb, diarrhea, Renal tubular acidosis, Gain of Acid - Diabetic ketoacidosis, Lactic acid. alcohol keto.
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| Causes of Met. Alkalosis | Increase in Base-Diuretic therapy, Loss of fixed acid- Severe vomitting, Naso suction
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