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Endocrine

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Question
Answer
Pituitary Acidophils & Basophils   FLAT PiG: Fsh, Lh, Acth, Tsh (Basophils) ProlactIn, Gh (Acidophils)  
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Alpha & Beta Subunits   - Alpha: Common to TSH, LH, FSH, hCG. Beta: Determines hormone specificity. - ACTH & MSH (and beta-endorphin) derived from POMC  
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Endocrine Pancreas Cells   Alpha cells: Glucagon, peripheral. Beta Cells: Insulin, central. Delta Cells: Somatostatin, interspersed.  
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TRH   -> TSH, Prolactin. Dopamine --| prolactin. Somatostatin --|TSH.  
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CRH   -> ACTH  
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Somatostatin (Octreotide) (physiologic & Therapeutic functions)   --| GH, TSH Octretoide treats gigantism, Acromegaly, Carcinoid, Gastrinoma, Glucagonoma (Necrolytic migratory erythema, stomatitis, chelosis)  
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POMC Derivatives   Produces Beta endorphin, ACTH, MSH  
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17alpha-Hydroxylase Deficiency   CAH. Aldosterone only, no Cortisol or Androgens. -> Hypertension & Hypokalemia. Phenotypic female w/o maturation.  
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21-Hydroxylase Deficiency   #1 CAH. Androgens only. No cortisol or aldosterone. -> Masculinization/female pseudohermaphrodite, Hypotension & hyperkalemia, elevated renin. Hypovolemic shock in newborn.  
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11beta-Hydroxylase Deficiency   CAH. Androgens & aldosterone pre-cursor (11-Deoxycorticosterone). No cortisol. -> Masculinization & HYPERtension.  
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PTH Function   - Activates Ca absorption (renal, GI) & inhibits PO4 absorption(renal). - Active 1,25(OH)2D production. - Osteoclast/blast stimulation.  
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Vit D Synthesis & Function   Sources: D2 (Plants) & D3(sun) –(liver)-> 25-(OH)D. Low PO4/Ca, High PTH: 25-(OH)D –(kidney)-> 1,25-(OH)2D. -> -> Ca & PO4 GI reabsorption & bone release.  
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Etiology of AlkPhos Elevation   Pagets, Osteoblastic bone tumor, HyperPTH, Rickets/Osteomalacia.  
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Glut4   Insulin-Dependent glucose uptake. Skeletal muscle & fat.  
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Glut1   Insulin-Independent glucose uptake. Brain, RBCs  
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cAMP-Mediated Hormones   hCG, Calcitonin, CRH, Glucagon, Straight-up “hormones”  
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cGMP-Mediated Hormones   Vasodilators (ANP, NO/Endothelium Derived Relaxing Factor)  
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IP3-Mediated Hormones   Hypothalamus-Derived Hormones (GhRH, TRH, GHRH, ADH, Oxytocin)  
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Steroid Receptor-Mediated Hormones   Glucocorticoids, Vit D, T3/4  
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Tyrosine Kinase-Mediated Hormones   Insulin, Growth FACTORS (IGF-1, FGF)  
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Cushings Syndrome Etiology   - Cushing Disease (Pituitary adenoma, only responds to high Dex suppression, others don’t respond) - Adrenal hyper/neoplasia - Small Cell Lung Cancer - Steroid use  
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Adrenal Insufficiency Sx, Etiology   - Primary Adrenal Insufficiency (Addison’s). HypoTN, skin hyperpigmentation (high ACTH). Tx: Glucocorticoids. - Waterhouse-Freidrichsen Syndrome (n. meningitidis bacteremia-> infarct). - Secondary: Pituitary insufficiency (low ACTH).  
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Pheochromocytomas Genetics & Characteristics   #1 adult adrenal tumor. VMA. MEN II, III (ret), neurofibromatosis. +/-Bilateral, malignant, ectopic, calcified, pediatric, inherited.  
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Neuroblastoma   #1 pediatric adrenal tumor. HVA. N-myc. Local or sympathetic chain-ectopic.  
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Sheehan’s Syndrome Cause & Sx   Postpartum hemorrhage/hypotn-> pituitary infarct. Poor lactation.  
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MEN I Cancers & Sx   Kidney stones & gastric ulcers. - Pancreas (ZE, VIPoma, Insulinoma) - Pituitary (Prolactinoma) - Parathyroid  
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MEN II Cancers   ret - Pheochromocytomas - (Para)Thyroid.  
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MEN III Cancers   ret. - Pheochromocytoma - Medullary thyroid carcinoma (Amyloid) - Oral/GI mucosal neuromas  
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Riedel’s Thyroiditis   Fibrosis of thyroid. HypoThyroid  
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Hashimoto’s Thyroiditis Sx, Dx   - Hypothyroid w/Thyrotoxicosis - AI: Anti-Microsomal & Thyroglobulin Antibodies. Hurthle Cells.  
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Subacute/de Quervain’s Thyroiditis Presentation & variants   Post-flulike illness. Jaw pain. - Granulomatous form is painful - Lymphocytic form is painless.  
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Toxic Multinodular goiter   TSH-independent TH production. Not malignant  
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Jod-Basedow Phenomenon   Thyrotoxicosis when endemic-goiter patient moves to Iodine non-deficient area.  
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Papillary Thyroid Carcinoma   #1, good prognosis. - Orphan annie nuclei, Psamomma bodies. - Increased risk w/childhood radiation  
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Follicular Thyroid Carcinoma   Good prognosis - uniform follicles.  
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Medullary Thyroid Carcinoma   Parafollicular C cells->calcitonin. - MEN II, III - Amyloid forming  
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Undifferentiated/Anaplastic Thyroid Cancer   Older patients, very poor prognosis  
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Thyroid Lymphoma   Hashimoto’s-associated  
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Acromegaly/Gigantism Dx, Tx   Dx’d w/ Poor glucose tolerance test. Octreotide tx Gigantism.  
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Primary HyperPTH Etiology, associations, labs.   - PTH Adenoma: elevated urine cAMP, calcuria, constipation, Alk Phos - Assoc. w/Osteitis Fibrosa Cystica (Bone cysts w/ brown fibrous tissue)  
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HypoPTH Etiology, PE   - DiGeorge - AI - Surgical - Chvostek + (facial nerve), Trousseau + (BP cuff-> carpal spasm) - PseudoHypoPTH: Kidney insensitivity to PTH. Short & small digits 4 & 5.  
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HyperCalcemia Etiology   CHIMPANZEES: - Calcium intake - HyperPTH, HyperThyroid - Iatrogenic (thiazides) - Multiple myeloma - Paget’s - Addison’s - Neoplasms - Z-E - Excess vit D, A - Sarcoid  
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DM1   Insulin dependent (profound hypoinsulinism). DKA common. Viral-triggered AI attack on beta cells. Weak gene association (hla-DR3,4)  
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DM2   Insulin Independent. Strong genetic association  
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DKA (Sx, Tx)   - Kussmaul respiration, N/V, abdominal pain - Anion gap metabolic acidosis. Intercellular hypoK (Arrhythmias), Mucormycosis, Rhizopus Infx, heart failure. - Rx: Fluid, insulin, K.  
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Diabetes Insipidus (Etiology, Dx)   - Pituitary tumor - trauma/surgery - Histiocytosis X - Secondary HyperCa [renal] - Lithium, Demeclocycloine - Dx: Water deprivation. Desmopressin (ADH analog) distinguishes central & nephrogenic. - Urine <1.006, blood >290 mOsm/L.  
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Diabetes Insipidus Tx   Central: Desmopressin. Nephrogenic: HCTZ, Amiloride, Indomethacin.  
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SIADH Etiology, Labs, Tx   - Small cell Lung Ca & Pulm Dz - CNS trauma - Cyclophosphamide - Labs: HypoNa, Urine > Serum osmolarity. - Tx: slowly w/ demeclocycline, Lithium (SE’s), H20 Restriction.  
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Carcinoid Syndrome Labs, Sx, Morphology, Tx   Elevated urine 5-HIAA. - GI enterochromaffin cell tumor (serotonin) outside GI. Sx: Flushing, Diarrhea, wheezing, peripheral edema. #1 appendix tumor, R Endocardial Fibrosis. MULTIPLE (metastatic, addt’l malignancy, multiple). - Tx: Octreotide  
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Insulin   Time: Lispro & Aspart < NPH < Lente < Ultralente. Tx DM1, HyperK  
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1st Gen Sulfonylureas   Tolbutamide, Chlorpropramide. SE: Disulfram-like effects.  
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2nd Gen Sulfonylureas   Glyburide, Gilmepiride, Glipizide.  
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Sulfonylurea mech   Close Beta cell K channel -> depolarization -> Ca influx -> Insulin release. Only DM2  
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Biguanides   Metformin Mech: Reduce gluconeogenesis, increase glycolysis, lowers serum glucose SE: Lactic Acidosis. CI:Renal, Liver Failure/Alcoholics, CHF, Sepsis  
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Glitazones   Pio/Rosi-glitazone Mech: Increase sensitivity to insulin via PPAR gamma. SE: Wt gain, edema, hepato & cardio toxic  
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Alpha Glucosidase Inhibitors   Acarbose & Miglitol Mech: inhibits brush border glucosidase -> slows glucose absorption SE: GI disturbances DM2 only  
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Methimazole, Propylthiouracil   Inhibits TH synthesis via -|thyroid peroxidase PTU also inhibits T4->T3. SE: Rash, Agranulocytosis & Aplastic Anemia  
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GH Therapy   GH Deficiency, Turner’s Syndrome  
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Desmopressin Therapy   Central DI  
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Glucocorticoid Use   Addison’s.  
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Secondary HyperPTH Labs, Associations   Renal failure: Low Ca, high PO4 & Alk Phs Assoc w/Renal osteodystrophy (bone lesions b/c high PTH)  
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Cortisol Physiology   Stimulates Gluconeogenesis, Glycolysis, Epinephrine synthesis, Proteolysis, Lipolysis, Neutrophil Demargination. Suppresses Insulin, non-neutrophil lymphocyte counts.  
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Neurophysin   Transports ADH & Oxytocin from Hypothalamus to Neurohypophysis  
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Acute Hyperthyroid Tx   Beta blockers  
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Kallmans Syndrome   GnRH migration failure-> primary hypoandrogenism. + Anosmia  
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Conn's Syndrome   Primary Aldosteronism: Adrenal Hyperplasia/Adenoma  
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