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Integrated CV Control

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Question
Answer
Medullary CV center   Vasomotor (ventrolateral medulla) Cardioacceleratory Cardioinhibitory  
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Blood Pressure Control   Baroreflex  
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High pressure receptors   Carotid sinus Aortic Arch  
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Low Pressure Receptors   Cardiopulmonary receptors pulmonary artery Junctino of atria w/ corresponding veins  
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Response to orthostatic pooling of blood   Decreased Stretch -> Increased SNS Decreased PNS  
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SNS on B1 in orthostatic hypotension   increased HR Increased contractility Increased SV  
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SNS on arterioles   Consticts Increased Peripheral Resistance  
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SNS on Veins   Increased Vascular Resistance Increased Stroke Volume Increased Cardiac Output  
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SNS on Kidneys   Increased Na Increased H2O Increased BV increased VR Increased CO  
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Baroreflex to increased arterial Pressure   NTS decreases SNS and Increases PNS resulting in decreased HR  
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Increased Stretch Leads to   increased HR VD to kidnes Decrease in ADH Myocytes release ANP  
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Decreased stretch leads to   no effect on HR Increased SNS to kidneys Increased release of ADH, AVP  
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Stretch of high-pressure receptors   Due to increased BP reflex works to decrease BP  
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Stretch of Low-pressure receptors   increased volume relex works to dump fluid  
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Hormonal Control   Epinephrine AVP ADH ANP  
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AVP or ADP   Vasopressin Anti-diuretic Hormone Synthesized in the hypothalamus and released from pituitary  
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Stimulus for release of vasopressin   Increased plasma osmolality Decreased volume Circulating Ang II  
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AVP Actions   vasoconstrict retention of fluid by kidney to increase volume  
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RAAS   Renin-Angiotensin-Aldosterone-System  
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Angiotension I ->   Angiotension II by enzyme ACE  
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Angiotensin II results in   Increased Aldosterone Decreased Na Excretion Increased Systemic Vascular Resistance  
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Role of chemoreceptors in AP regulation   Important role in severe hypoxia Decreased pO2, Increased pCO2, and Decreased pH Central Sense: elevated CO2 leading to decreased pH  
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Created by: tjamrose
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