| Question |
 |
|
| Answer |
|
|
| Medullary CV center |
Vasomotor (ventrolateral medulla)
Cardioacceleratory
Cardioinhibitory |
| Blood Pressure Control |
Baroreflex |
| High pressure receptors |
Carotid sinus
Aortic Arch |
| Low Pressure Receptors |
Cardiopulmonary receptors
pulmonary artery
Junctino of atria w/ corresponding veins |
| Response to orthostatic pooling of blood |
Decreased Stretch ->
Increased SNS
Decreased PNS |
| SNS on B1 in orthostatic hypotension |
increased HR
Increased contractility
Increased SV |
| SNS on arterioles |
Consticts
Increased Peripheral Resistance |
| SNS on Veins |
Increased Vascular Resistance
Increased Stroke Volume
Increased Cardiac Output |
| SNS on Kidneys |
Increased Na
Increased H2O
Increased BV
increased VR
Increased CO |
| Baroreflex to increased arterial Pressure |
NTS decreases SNS and Increases PNS resulting in decreased HR |
| Increased Stretch Leads to |
increased HR
VD to kidnes
Decrease in ADH
Myocytes release ANP |
| Decreased stretch leads to |
no effect on HR
Increased SNS to kidneys
Increased release of ADH, AVP |
| Stretch of high-pressure receptors |
Due to increased BP
reflex works to decrease BP |
| Stretch of Low-pressure receptors |
increased volume
relex works to dump fluid |
| Hormonal Control |
Epinephrine
AVP
ADH
ANP |
| AVP or ADP |
Vasopressin
Anti-diuretic Hormone
Synthesized in the hypothalamus and released from pituitary
|
| Stimulus for release of vasopressin |
Increased plasma osmolality
Decreased volume
Circulating Ang II |
| AVP Actions |
vasoconstrict
retention of fluid by kidney to increase volume |
| RAAS |
Renin-Angiotensin-Aldosterone-System |
| Angiotension I -> |
Angiotension II by enzyme ACE |
| Angiotensin II results in |
Increased Aldosterone
Decreased Na Excretion
Increased Systemic Vascular Resistance |
| Role of chemoreceptors in AP regulation |
Important role in severe hypoxia
Decreased pO2, Increased pCO2, and Decreased pH
Central Sense: elevated CO2 leading to decreased pH |
mr. charles11
