normal indicies and drug review
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| CO: | vol/min
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| CO= | 4-8 L/min
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| CI: | CO/BSA
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| CI= | 2.5-5.0 L/min/m2
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| MAP: | [LVP + 2 (RAP)]/3
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| MAP= | 80-100 mmHg
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| MPAP: | [RVP + 2 (LAP)]/3
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| MPAP= | 10-20 mmHg
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| UO= | 60 mL/hr
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| CVP: | RAP, reflecting total vol.
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| CVP= | 2-6 mmHg
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| PCWP: | LAP=LVEDP
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| PCWP= | 4-12 mmHg
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| SVR: | [(MAP-CVP)*80]/CO
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| SVR= | 900-1400 dynes x s x cm5-
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| PVR: | { (PAP-CVP)* 80 } / (CO)
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| PVR= | 150-250 dynes x s x cm-5
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| MC RMP= | -80 to -90 mV
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| affect of inhibiting Na+ influx into MC: | Phase 0 less steap/high, increased refractory time, decreased HR
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| Phase of MC AP involving Mg: | Phase 4 for function of NaK pump restoring ion balence
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| SA node rate= | 60-100
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| AV junction rate= | 40-60
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| Ventricle rate= | <40
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| Absolute Refract: | P to mid T
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| Relative refract: | Mid to end T
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| Intropin 1-2 ug/kg/min: | promote renal, mesenteric blood flow? not
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| Intropin 2-10 ug/kg/min: | inotropic effects
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| Intropin >10 ug/kg/min: | vasopressor (constriction)
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| Dig FX | +contractility, -dromotropic
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| treat PACs: | verapamil, diltiazem
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| treat Afib/flut: | Ca++/B/Dig,Procainamide
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| treat SVT/PSVT: | adenosine, Ca/B/K block
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| treat Vtach/fib: | Epi/vasopressin, lidocaine, amiodorone, MgSO4, procain
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| I AA mechanism: | block influx of Na into both MC (phase 0) and PM cell (phase 4)... - conductivity and + refractory time; - automaticity (- HR)
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| II AA mechanism: | Block the B1 receptors
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| III AA mechanism: | slow efflux of K+ out MC, so repolarization +; +absolute refractory time; -conduction; -automaticity (- K out of cell, -phase 4 steepness
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| IV mechanism: | -Ca influx (-HR), prevents SM contraction, vascular relaxation
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| adenosine mechanism: | stimA1,-AV cond,-HR, +vasoD
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| MgSO4 mechanism: | return ion balence esp. during CPR
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| treat acute HT | Nipride
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| treat + precap arteriole R | Nipride, Adalat, Capoten, Vasotec
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| treat + precap venule R | Morphine
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| Persantine FX | (dypyrimidamole) inhibits platelet aggregation
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| Cardiazem FX | (diltiazem) -Mi02, -HR, vasoD
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