Stack #121551

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ODC shift left   More alkalosis, less DPG, hemoglobin has more affinity for O2, P50 decreases, Hgb is more saturated at a given PO2.  
ODC shift right   More acidic, more DPG, hemoglobin has less affinity for O2, P50 increases, Hgb is less saturated at a given PO2.  
Normal P50   27mmHg  
CO2 diffusion rate   20 times faster than O2  
Dead space   anatomic 1ml per pound, alveolar dead space can not be calculated, physiological dead space the sum of alveolar and anatomical deadspace.  
Anatomic Shunt (true shunt)   The portion of cardiac output that enters the left side of the heart without comming in contact with an alveolus for gas exchange.  
Causes of Anatomic Shunts   Congenital heart disease, Intrapulmonary fistula, Vascular tumors.  
Capillary Shunt (true shunt)   No gas exchange at alveoli.  
Absolute shunt   When more O2 doesn't help/ refractory to oxygen therapy.  
Shunt-like effect   When pulmonary capillary perfusion is in excess of alveolar ventilation.  
Causes of Capillary Shunts   Atelectasis, alveolar fluid accumulation, or consolidation.  
% of total CO2 transported to the lungs in RBC's by bicarbonate (HCO3)   63%  
The Haldane Effect   Deoxygenated blood enhances loading of CO2 and oxyegnated blood enhaces the offload of CO2.  
The Bohr Effect   The effect of PCO2 and PH on the oxyhemoglobin curve.  
Anatomic Shunt normals   2 to 5%  
Things that move ODC right   Temp increase, PCO2 increase, PH decrease (more H+), DPG increase.  


   

 
 

 
 

 

 
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