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Cards ECG


Diffuse ST elevation in most leads, peaked T waves in V leads = pericarditis
Irregular irregular Atrial fibrillation; (if > 48 or chronic: anticoagulate)
EKG arrhythmia assoc w/COPD multifocal atrial tachy
LVH on EKG S in V1 + R in V5-V6 > 35; aVL (R) > 11; LAD; wide QRS; ST/TW changes
RVH on EKG RAD; V1: R>S; R gets smaller V1 -> V6; S wave V5-V6; wide QRS
tachy on EKG (regular/narrow) = sinus; atrial tachy or flutter; re-entrant (AVNRT/PSVT)(usu after p wave); give adenosine
tachy on EKG (irreg/narrow) = sinus tach w/PAC; MAT; A fib; atrial flutter w/variable block
tachy on EKG (regular/wide) = V-tach; SVT w/aberrancy (BBB); SVT w/WPW; pacemaker tachy
tachy on EKG (irreg/wide) = V-fib; torsades; irreg SVT w/aberrancy; irreg SVT w/WPW
RBBB on EKG V1-V2: RSR'; slurred S wave at I, aVL, V5-V6; biphasic QRS at I
LBBB on EKG QRS >120; notched/slurred R in I, aVL, V5-V6; teepee (big pos R) V5-V6, no Q waves in same; ST & T usually opp direction of QRS (=ischemia) (occ QRS-T concordance); V1-V2: broad negative rS or QS
Hypercalcemia on EKG Shortened QT, Wide QRS, absent ST segment
Hypocalcemia on EKG Prolonged QT in II, V1, and V5, predisposition to V-tach
Hyperkalemia on EKG short QT, wide QRS, flat P wave, peaked T waves
Hypokalemia on EKG U waves, ST flattening, TWI, ST depression; areflexia, paralysis, ortho hypotension, ileus
Hypomagnesemia on EKG prolonged PR & QT, and wide QRS
Electrolyte imbalance: Shortened QT, Wide QRS, absent ST segment = hypercalcemia
Lyte imbalance: Prolonged QT in V1, II, and V5, predisposition to V-tach = hypocalcemia
Electrolyte imbalance: short QT, wide QRS, peaked T waves = Hyperkalemia
Electrolyte imbalance: U waves, ST flattening, TWI, ST depression; areflexia, paralysis, ortho hypotension, ileus = Hypokalemia
Antiarrhythmic: pos inotrope, neg chronotrope, decreases conduction velocity thru AV node = digoxin
Effects of CCB: Class IV, vasodilator; neg chronotrope, neg inotrope
Effects of beta blockers: Class II; neg chronotrope, neg inotrope
COPD on EKG Right axis deviation (RAD = RAD)
S1 Q3 TIII = Large S wave (I), ST depression (II), Q wave (III) = PE
Hypermagnesemia on EKG: wide QRS, long PR & QT
Short PR, wide QRS, Delta wave Wolf-parkinson-white; avoid Digoxin
Most common cause of tachyarrhythmia reentry; >1 pathway
Inferior STEMI reciprocates to which leads? AVL and I
Anterior STEMI reciprocates to which leads? Inferior leads (II, III, AVF)
Lateral STEMI reciprocates to which leads? Inferior leads (II, III, AVF)
What type of STEMI reciprocates to the anterior leads? None
Inferior MI: ECG correlation MOST COMMON MI. (II, III, aVF); RCA; left circumflex if left-dominant
Anterior MI: ECG correlation V1-V5; LAD
Lateral MI: ECG correlation I, aVL, V5-V6; Circumflex
Inferolateral MI: ECG correlation II, III, aVF, I, aVL; large RCA, or left-dominant Left circumflex
Septal / Posterior MI: ECG correlation V1-V2; LCA (or septal branch of LAD)
EKG changes, N/V, yellow-green visual disturbances = Digoxin toxicity (Hypokalemia will make worse)
Q waves in an MI usually develop within: 12-36 hours
MVP on ECG: often normal; ST depression or TWI in III & aVF
HCM on ECG = LVH, nonspecific ST-T abnormalities, deep septal Q waves in inferior leads, or tall narrow R waves in V1-V2
VSD on ECG LVH, RVH, atrial enlargement
ASD on ECG: RAD, rsR' pattern; RVH -> RBBB
Alcoholic with palpitations, arrhythmia = Atrial fibrillation (Holiday heart)
Created by: Adam Barnard Adam Barnard