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MED SURG TEST 3

QuestionAnswer
Cardiomyopathy Ejection Fraction Usually less than 50%
Dilated Cardiomyopathy (Congested) Most Common, charcterized by cardiomegaly (not much thickening and hypertrophy)
Causes of Dilated Cardiomyopathy Pregnancy, Viral Infections
Manifistations of Dilated Cardiomyopathy Exercise intolerant, dry cough, dyspnea, palpitations, JVD< Anorexia, Extra Heart Sounds, Pulmonary Crackles, Edema, Pallor, Enlarged Liver
Cardiac Catheterization Can determine pressure readings and ejection fraction
Endomyocardial Biopsy May be used for diagnosis of cardiomyopathy. Will determine if tissue is damaged or not
Treatment for Dilated Cardiomyopathy Similar as to CHF- control the damage to the heart- May need transplant
Implantable Cardoverter Defibrillator Used for Low Ejection Fraction
Hypertrophic Cardiomyopathy Walls and Septum Getting thicker, so no dilation of ventricle. Enlargement can obstruct blood flow
Hypertrophic Cardiomyopathy S/S Maybe asymptomatic, or have dyspnes, fatigue, angina, syncope, and arrhythmias. PMI Moved down and lateral
Restrictive CMP Least Common- damaged tissue that won't stretch- so requires high diastolic pressures
Hypertrophic CMP Tx Trim off extra heart muscle or alcohol ablation
Alcohol Ablation Destorys some of tissue to allow more room in ventricle
Manifestations of Endocarditis Fever and Chills, Tachycardia, SOB, Clot Symptoms, Joint Pain, Osler's Nodes, Janeway Lesions, Roth's Spots, New Murmurs, CHF, Splinter Hemorrhages
Osler's Nodes split pea-tender-transient on toes, fingers, palms, and soles
Janeway's Lesions Nontender and hemorrhagic on soles and palms
Roth's Spots Retinal Hemorrhages- rare and longitudinal
Treatment of Infective Endocarditis Antibiotics, Antipyretics, rest, Repeat blood cultures, surgical repain
High Risk Patients of Infective Endocarditis artificial valves or history of endocarditis
Moderate Risk Patients of Infective Endocarditis Mitral Valve Prolapse
Acute Pericarditis Fluid between layers increases pressure and restricts heart (may cause cardiac tamponade)
Causes of Pericarditis Infection, Heart Attack, Cancer spreading, Radiation Treatment, Injury or Surgery, Drugs, Rheumatoid Arthiritis
Addresslers Syndrome Pericarditis resulting from a heart attack a month after the heart attack
Clinical Manifestation of Pericarditis Sharp Pain (worsens on deep breath), Friction Rub, Fever, Pain when swallowing
Complications of Pericarditis Pericardial Effusion and Cardiac Tamponade
Beck's Triad Seen in Cardiac Tamponade: JVD, Tachycardia, and Muffled Heart Sounds
Pulses Parodoxis Decrease in systolic on inspiration > 10 mmHg (cardiac tamponade)
S/S pericardial Effusion SOB, cough, Increased Respirations, S1 S2 muffled
Pain relief of pericarditis Eased by sitting and leaning forward
Ischemia ECG change localized ST elevation
Pericarditis ECG change widespread changes in ST segement, T wave, and QR interval
Constrictive Pericarditis Scarring of pericardial sac Similar to CHF symptoms
Pericardial Knock Sign of Constrictive Pericarditis, auscultated on LSB
ECG Changes in Contrictive Pericarditis Flattening of complexes
Myocarditis Inflammation of heart muscle- no treatment- support heart
Casues of Myocarditis Viral
Clinical Manifestation of Myocarditis Fever, Fatigue, Sore Throat, Dyspnea, N&V, JVD, Extra Heart Sounds, CHF
Diagnoses of Myocarditis ECK nonspecific, WBC may be increased, Endomyocardial Biopsy
Rheumatic Fever Imflammation of all three heart layers- scars valves. usually begins as sore throat
Pancardits In rheumatic fever- all layers of heart and the valves vegetate
Aschoff's Bodies Lesions on myocardium of swollen collagen seen in Rheumatic Fever (can be in Lungs, Joints, and Brain)
Major Criteria for Rheumatic Fever Carditis, Chorea, Nodules at joints, Rach of Chest, back, and ABD
Minor Criteria for Rheumatic Fever Fever,HX of Rheumatic Heart Disease, Lab Findings, Prolonged PR interval
Erythrocyte Sedimentation Rate in Rheumatic HD Increases with inflammation
C-Reactive Protein during Rheumatic HD increases during systemic infection
Major Complication of Rheumatic Fever Chronic Rheumatic Carditis (cordae tendinae damaged)and valve structure changed (mostly mitral)
Mitral Valve Stenosis Causes Rheumatic Heart Disease, Rheumatoid Arthiritis, Lupus, and Genetic
Mitral Valve Stenosis Left Atrial Pressure is higher than Left Ventricle= lungs compression
Clinical Manifestations of Mitral Valve Stenosis Dyspnea, Palpitations, Hoarsness(caused by A Fib), Emboli, S1 Snap
Most frequent cause of Mitral Valve Regurgitation MI (enlarged left ventricle and atrium)
Acute Mitral Valve Regurgitation Pulmonary edema, shock (decreased BP)
Chronic Mitral Valve Regurgitation Ventricular Failure- weakness and fatigue, SOB< peripheral edema, S3, murmur in apex
Mitral Valve Prolapse Murmur, Clicks, PVCs and Tachycardias, Chest pain not responsive to nitrates, SOB, palpitations, syncope, Risk for infective endocarditis- May have Marffans.
Aortic Valve Stenosis Causes Congenital Heart Disease, Rheumatic Heart Disease, and Degenerative Heart Disease
Symptoms of Aortic Valve Stenosis Angina, Syncope,Abnormal Heart Sounds and heart Failure- NITROGLYCERIN CONTRAINDICATED
Causes of Aortic Valve Regu
Cardiomyopathy Ejection Fraction Usually less than 50%
Dilated Cardiomyopathy (Congested) Most Common, charcterized by cardiomegaly (not much thickening and hypertrophy)
Causes of Dilated Cardiomyopathy Pregnancy, Viral Infections
Manifistations of Dilated Cardiomyopathy Exercise intolerant, dry cough, dyspnea, palpitations, JVD< Anorexia, Extra Heart Sounds, Pulmonary Crackles, Edema, Pallor, Enlarged Liver
Cardiac Catheterization Can determine pressure readings and ejection fraction
Endomyocardial Biopsy May be used for diagnosis of cardiomyopathy. Will determine if tissue is damaged or not
Treatment for Dilated Cardiomyopathy Similar as to CHF- control the damage to the heart- May need transplant
Implantable Cardoverter Defibrillator Used for Low Ejection Fraction
Hypertrophic Cardiomyopathy Walls and Septum Getting thicker, so no dilation of ventricle. Enlargement can obstruct blood flow
Hypertrophic Cardiomyopathy S/S Maybe asymptomatic, or have dyspnes, fatigue, angina, syncope, and arrhythmias. PMI Moved down and lateral
Restrictive CMP Least Common- damaged tissue that won't stretch- so requires high diastolic pressures
Hypertrophic CMP Tx Trim off extra heart muscle or alcohol ablation
Alcohol Ablation Destorys some of tissue to allow more room in ventricle
Manifestations of Endocarditis Fever and Chills, Tachycardia, SOB, Clot Symptoms, Joint Pain, Osler's Nodes, Janeway Lesions, Roth's Spots, New Murmurs, CHF, Splinter Hemorrhages
Osler's Nodes split pea-tender-transient on toes, fingers, palms, and soles
Janeway's Lesions Nontender and hemorrhagic on soles and palms
Roth's Spots Retinal Hemorrhages- rare and longitudinal
Treatment of Infective Endocarditis Antibiotics, Antipyretics, rest, Repeat blood cultures, surgical repain
High Risk Patients of Infective Endocarditis artificial valves or history of endocarditis
Moderate Risk Patients of Infective Endocarditis Mitral Valve Prolapse
Acute Pericarditis Fluid between layers increases pressure and restricts heart (may cause cardiac tamponade)
Causes of Pericarditis Infection, Heart Attack, Cancer spreading, Radiation Treatment, Injury or Surgery, Drugs, Rheumatoid Arthiritis
Addresslers Syndrome Pericarditis resulting from a heart attack a month after the heart attack
Clinical Manifestation of Pericarditis Sharp Pain (worsens on deep breath), Friction Rub, Fever, Pain when swallowing
Complications of Pericarditis Pericardial Effusion and Cardiac Tamponade
Beck's Triad Seen in Cardiac Tamponade: JVD, Tachycardia, and Muffled Heart Sounds
Pulses Parodoxis Decrease in systolic on inspiration > 10 mmHg (cardiac tamponade)
S/S pericardial Effusion SOB, cough, Increased Respirations, S1 S2 muffled
Pain relief of pericarditis Eased by sitting and leaning forward
Ischemia ECG change localized ST elevation
Pericarditis ECG change widespread changes in ST segement, T wave, and QR interval
Constrictive Pericarditis Scarring of pericardial sac Similar to CHF symptoms
Pericardial Knock Sign of Constrictive Pericarditis, auscultated on LSB
ECG Changes in Contrictive Pericarditis Flattening of complexes
Myocarditis Inflammation of heart muscle- no treatment- support heart
Casues of Myocarditis Viral
Clinical Manifestation of Myocarditis Fever, Fatigue, Sore Throat, Dyspnea, N&V, JVD, Extra Heart Sounds, CHF
Diagnoses of Myocarditis ECK nonspecific, WBC may be increased, Endomyocardial Biopsy
Rheumatic Fever Imflammation of all three heart layers- scars valves. usually begins as sore throat
Pancardits In rheumatic fever- all layers of heart and the valves vegetate
Aschoff's Bodies Lesions on myocardium of swollen collagen seen in Rheumatic Fever (can be in Lungs, Joints, and Brain)
Major Criteria for Rheumatic Fever Carditis, Chorea, Nodules at joints, Rach of Chest, back, and ABD
Minor Criteria for Rheumatic Fever Fever,HX of Rheumatic Heart Disease, Lab Findings, Prolonged PR interval
Erythrocyte Sedimentation Rate in Rheumatic HD Increases with inflammation
C-Reactive Protein during Rheumatic HD increases during systemic infection
Major Complication of Rheumatic Fever Chronic Rheumatic Carditis (cordae tendinae damaged)and valve structure changed (mostly mitral)
Mitral Valve Stenosis Causes Rheumatic Heart Disease, Rheumatoid Arthiritis, Lupus, and Genetic
Mitral Valve Stenosis Left Atrial Pressure is higher than Left Ventricle= lungs compression
Clinical Manifestations of Mitral Valve Stenosis Dyspnea, Palpitations, Hoarsness(caused by A Fib), Emboli, S1 Snap
Most frequent cause of Mitral Valve Regurgitation MI (enlarged left ventricle and atrium)
Acute Mitral Valve Regurgitation Pulmonary edema, shock (decreased BP)
Chronic Mitral Valve Regurgitation Ventricular Failure- weakness and fatigue, SOB< peripheral edema, S3, murmur in apex
Mitral Valve Prolapse Murmur, Clicks, PVCs and Tachycardias, Chest pain not responsive to nitrates, SOB, palpitations, syncope, Risk for infective endocarditis- May have Marffans.
Aortic Valve Stenosis Causes Congenital Heart Disease, Rheumatic Heart Disease, and Degenerative Heart Disease
Symptoms of Aortic Valve Stenosis Angina, Syncope,Abnormal Heart Sounds and heart Failure- NITROGLYCERIN CONTRAINDICATED
Causes of Aortic Valve Reguurgitation Rheumatic Fever, Congenitive, or Syphillis
Backwords blood flow in Aortic Valve Regurgitation Decreased Cardiac Output, dilated ventricle, and decreased contractibility
S/S of aortic valve regurgitation Weakness, dyspneas, hypotension, water-hammer pulse, austin-flint murmur, soft s1; s3 and s4 present
Austin-Flint Murmur Apex. Mitral Valve getting hit from aorta and atrium at same time
Tricuspid Regurgitation Overload to right side of heart and results from Pulmonary HTN or right ventricular dysfunction
P wave Represents SA node stimulation and atrial depolarization
Causes of abnormal P waves Atrial Hypertrophy - Increased amplitude or width
PR Interval Time impulse to tavel from Atria to AV node- Norm: 0.12-0.2
Prolonge PR interval indications diseased AV node, Ischemia, drug effects, or increased vagal tone
QRS Complex Ventricular depoloarization- Norm: 0.06-0.10
QRS > 0.12 intraventricular conduction delay- may be caused by bundle branch block, WPW syndrome, and hyperkalemia
ST Segment On isoelectric line- represents initial phase of ventricular repolarization
Prolonged ST Segement cause Hypocalcemia
Elevated ST Segment Pericarditis, MI, or left ventricular aneurysm
Depressed ST Segment Subendocardial ischemia, electrolyte disturbance, drug effect
T Wave Ventricular Repolarization Completed
Inverted T Wave Infarctions, Ischemia, Injury, Hypertrophy
Tall, Peaked T Wave Hyperkalemia or acute injury
U wave Not normally present, may be present in hypokalemia
QT interval complete duratiion of ventricular depolarization and repolarization Norm: 0.20-0.40
Prolonged Qts antiarrhythmic drugs (amiotarone, allegra, and claritis
Complication of prolonged QT polymorphic V Tach (torsades de pointes)
Causes of Shortened QT digitalis and tachycardia
Sinus Tach Hemodynamic Consequence Decreased SV= Decreased CO due to shortened diastole
Respiratory Reflex R-R interavl shortens during inspiration due to increased venous return at this point
Treatment of sinus bradycardia Underlying cause treated- ACLS-atropine
Treatment of Sinus Tachycardia Underlying cause treated, beta blockers
Causes of SA block, pauses, and arrest Increased vagal stimulation (suctioning), MI, Myocarditis, drug effects (digitalis)
Causes of PACs Stimulants, hypoxia, digitalis, enlarged atria
Treatment of Atrial Tackycardia Vagal/Valsalvar manuever, carotid massage, adenosine IV, cardizem bolus, synchronoized cardioversion
SVT Supraventricular Tachycardias
Wide QRS Ventricular
Narrow QRS Atrial Tachycardia
Paroxysmal Tach Sudden onset or cessation
Atrial Fib Chaotic with NO p waves
A FIB- if ventricular rate become regular Suspect digitaliz toxicity
Junctional Escape Rhythm AV node pacing at 40-60 bpm
Treatment of Junctional Escape Rhythm usually none- atropine, TCP, d/c caustive drug
Premature Ventricular Contractions Wide QRS (maybe double R's) Bigeminy, trigeminy, couplet,
V TACH Three or more PVCs in a row
Causes of PVC ischemia, hypoxia, acidosis, electrolyte imbalance
Treatment of PVC not usually treated- correct electrolytes, optimize O2, d/c causative drug, administer amiodarone or lidocaine
Treatment of V-Tach VT with pulse- antidysrhythmias Pulseless VT- immediate debibrillation
Shock during VTACH Never On T wave. only on QRS
Polymorphic V Tach Torsades de pointes- irregular and wide QRS (hurricane effect)
Rate of Torsades de pointes 150-250 bpm
Causes of Torsades de pointes hypo- mag and hypo-kalemia, antiarrhythmic drug therapy
Treatments of Torsades de pointes MgSO4
VFIB NO pulse, No BP
VFIB Treatment Immediate Debrillation followed with antiarrhythmics
Ventricular Asystole No ventricular Activity- may still see P wave
V-Asystole Treatment Epinephrine- No point in defibrilation- usually fatal
Hypokalemia ECG flattened and inverted T wave, U Wave, and ST depression
Hyperkalemia ECG Tall T waves, Widened QRS, Prolonged PR, Flat P waves
PVC causes Hypokalemia and Hypoxemia
Cough CPR Unstable clint in V Tach
Vagal Maneuvers Used for Supraventricular Tachydysrhythmias
Cardioversion Lower version of defib used for tachycardias- synchronized with R wave
Defibrillation Used for pulseless V Tach and VFIB
Stable Angina Exertional
Unstable Angina Preinfaction angine- pain not relieved with nitroglycerin
Variant Angina Prinzmetal's or Vasospastic Angina- May occur at rest
Intractable angina Chronic, incapactitation, and unresponsive to interventions
Myocarditis Causes Pericaditis, systemic infection, allergic response
Complications of Myocarditis Thrombus, heart failure, and cardiomyopathy
Endocarditis Risk Factors IV drug users, valve replacements, mitral valve prolapse
Harch Systolic Crescendo-decrescendo Murmyr Aortic Stenosis
Blowing decrescendo diastolic murmur Aortic Insufficiency
Tricuspic Stenosis S/S Fluttering Sensation at neck due to obstructed venous flow, Cyanosis, Rumbling diastolic murmur
Fusiform aneurysms diffuse dilation that involves the entire circumference of the arterial segment
Saccular aneurysms localized outpouching of artery wall
Dissecting Aneurysms blood seperates the layers of the artery wall forming a cavity between them
Thoracis Aneurysm Pain Neck shoulders lower back and ABD
ABD aneurysm Pain in abd or lower back
S/S Rupturing Aneurysm Severe ABD and back pain, lumbar fain radiating to flank and growin, Hypotension, increased pulse, signs of shock, hematoma at flank
Idioventricular Rhythm AV NODE Response: Absent P wave Widened QRS > 0.12 sec. Also called " dying heart" rhythm Pacemaker will most likely be needed to re-establish a normal heart rate.
Virchow's Triad Affect Venous Thrombosis Development: Endotherlial injury, Venous Stasis, Hypercoagulable blood
Valve Cusps Common site of thrombus formation
DVT Treatment Bedrest, Elevation of extremity, anticoagulants
Risk Factors for PAD Smoking, Hyperlipidemia, Hypertension, Diabetes
Inlet Flow Trunk More damage
Outlet Flow Legs- less tissue damage
PAD: Stage I No pain, begin having build ups; pulses present
PAD: Stage II Intermittent Claudication
PAD: Stage III Worsened IV, Rest Pain- hang feet off bed
PAD: Stage IV Necrotic Tisses
Common Sites for PAD Illiac, Femoral, Renal
Most common site for diabets for PAD arteries below knees
Complications of PAD Atrophy, non-healing ischemic ulcers, amputations
Diagnostics for PAD Ankle Brachial Index (perferred 1 +)
Antherotomy Similar to heart cath- but shaves plaques and vacuums
Endarterectomy Blockage and vessel is cut open and plaques removed then sewn back up- USUALLY IN CAROTID
Signs and Symptoms of Aneurysm Rupture Severe back pain, shock signs, blue toe syndrome, and grey turner's sign
Blud Toe Syndrom Shower of clots moves to extremities
Grey Turner's ABD bleed- skin appears grey
NU interventions for endovascular graft keep still to avoid leaking, BP in narrow range
Complications of Dissection Aneurysm Cardiac Tamponade
Goal of Aneurysm Care (priority) Decrease BP and cardiac contractility
First Line Meds for Aneurysm Vasodilators and Beta Blockers
Created by: mcheflin
 

 



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