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-zosin alpha adrenergic blocker
-lol beta adrenergic blocker
-zepam, -zolam BZO
-pril ACEI
-sartan angiotensin II receptor blocker
-dipine Ca channel blocker
-statin LDL-lowering HMG-CoA reductase inhibitor
-sone, -olone corticosteroid
-parin anticoagulant
-prazole PPI
-terol, -phylline bronchodilator
-semide loop diuretic
How does alpha-2 stimulation differ from other sympathetic receptor sites? Can inhibit release of epinephrine or NE from presynaptic neuron
Adrenergics, sympathomimetics, adrenergic agonists mimic the effects of? NE
Sympatholytics, adrenergic blockers, and adrenergic antagonists block the effects of NE
Cholinergics, parasympathomimetics, cholinergic agonists, cholinomimmetics mimic the effects of Ach
Anticholinergics, parasympatholytics, and cholinergic antagonists block the effects of Ach
Sx of sulfite sensitivity Dizziness, loss of consciousness, hypotension, syncope, clammy flushed skin, pruritus, cyanosis, bronchospasm, wheezing, SOB, laryngeal edema, respiratory arrest Epinephrine, NE, phenylephrine, methoxamine, dopamine, dobutamine, metaraminol, fenoldopam
Pt edu for pts receiving clonidine (Catapres) Never stop abruptly- can cause rebound HTN
How does dopamine (Intropin) affect renal hemodynamics? High doses (bind alpha-1 receptors) cause vasoconstriction in kidneys, can cause renal failure. Low doses (binds dopaminergic receptors) causes increased GFR, renal blood flow, and sodium excretion (overall, increased renal output)
Why is dopamine (Intropin) and fenoldopam (Corlopam) only admined in acute care envts? Need to closely monitor BP, CO, urine output, pulmonary wedge pressure b/c can cause severe dysrhythmias and vasoconstriction (dopamine) or acute cerebral infarction or hemorrhage (fenoldopam)
AE of prazosin (Minipress) 1st dose syncope
Why are beta blockers used cautiously in pts with resp disorders? Can cause development of obstructive pulmonary disorders
Cholinergic crisis overstimulation at nm junction due to too much Ach, give atropine
Myasthenic crisis lack of Ach due to undermedication, give more prostigmin can cause inability to speak, breathe, or swallow in pts with myasthenia gravis
Common AEs of atropine blurred vision, urinary retention, constipation, dry mouth
Location of beta 2 receptors lungs
location of beta 1 receptors heart, kidneys
location of alpha 1 receptors prostate, peripheral vasculature
location of alpha 2 receptors terminal end of presynaptic neuron
propranolol (Inderal) effects on blood glucose None, but inhibits perception of hypoglycemic sx
Body SA sqrt [weight (kg) x height (cm)]/3600
-triptans serotonin agonists
LFTs bilirubin, AST, ALT, alk phosphatase, PTT, aPTT
renal function tests BUN, serum creatinine, creatinine clearance
hematopoietic function tests CBC- Hgb, Hct, WBC, platelets
6 rights of drug admin Right: patient, time, drug, dose, route, documentation
IM sites deltoid, rectus femoris, vastus lateralis, dorsogluteal, ventrogluteal
half-life time required to remove 50% of the blood [drug]
steady state 4-5 half lives, admin rate=excretion rate
Created by: lmr24