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OPT Periapical

mechanical, thermal, chemical and bacterial 4 types of noxious stimuli on pulp
reversible pulpitis tissue can return to health if stimuli removed, short duration of pain
irreversible pulpitis plural damage beyond point of recovery; severe, lingering, and spontaneous pain
chronic hyperplastic pulpitis (pulp polyp) in young people, pulpits produces granulation tissue that extrudes from the pulp chamber
secondary dentin dentin formed after completion of the crown
primary dentin dentin formed before crown completion
physiologic secondary dentin normal deposition of dentin that shrinks the chambers due to regular aging
reparative secondary dentin (irregular/tertiary) local dentin laid down in response to focal injury
calcific metamorphosis trauma leads to early obliteration of the pulp chamber; yellow clinical crown
periapical granuloma (chronic apical periodontitis) inflamed granulation tissue at the apex of nonvital teeth
fibrous (periapical) scar defect after periapical granuloma that heals with collagen instead of bone
periapical cyst (radicular cyst, apical periodontal cyst) epithelial-lined cavity stimulated by inflammation; looks identical to granuloma on radiographs
residual periapical cyst inflammatory material that is left after an extraction that causes inflammatory cyst
lateral radicular cyst inflammatory cyst next to a root secondary to pulpal infection or periodontal disease
periapical abscess accumulation of acute inflammatory cells at apex of nonvital teeth
phoenix abscess acute exacerbation of a chronic periapical lesion
osteomyelitis infection caused by involvement of adjacent marrow spaces
cellulitis extension of abscess to overlying soft tissue
cutaneous sinus purulent material that drains through the skin
parulis (gum boil) mass of granulation tissue at opening of sinus tract in the oral cavity
osteomyelitis inflammatory process in medullary spaces or cortical bone that is away from the site that is initially infected
acute osteomyelitis occurs when acute inflammatory process spreads through medullary bone spaces; usually short duration
sequestrum necrotic bone fragments
involucrum necrotic bone encased by vital bone
chronic osteomyelitis occurs secondary to defensive response producing granulation tissue that forms a scar in attempt to wall off the infection; requires surgery to remove infected material
diffuse sclerosing osteomyelitis when infectious process directly responsible for bone sclerosis; related to {condensing osteitis}
primary chronic osteomyelitis chronic pain, multifocal bone sclerosis without infection; includes {chronic recurrent multifocal osteomyelitis, CRMO} and {synovitis, acne, pustulosis, hyperostosis, and osteitis, SAPHO}
chronic tendoperiostitis reaction of bone to overuse of masticatory muscles, notching of mandible
condensing osteitis focal areas of bone sclerosis associated with apices of teeth with pulpitis
bone scar residual area of condensing osteitis after resolution of inflammation
proliferative periostitis (periostitis ossificans) periosteal hyperplasia secondary to adjacent inflammation aka Garre osteomyelitis; several rows of reactive bone that expand to onionskin pattern in juveniles
alveolar osteitis (dry socket, fibrinolytic alveolitis) destruction fo initial clot prevents healing after extraction
Created by: Alexandra Arnold Alexandra Arnold